Basic mechanisms leading to focal emphysema in coal workers' pneumoconiosis

Coal miners develop focal emphysema characterized by dilatation of second- and third-order respiratory bronchioles with coal mine dust-laden macrophages infiltrating the wall. A reticulin network with small amounts of collagen and atrophy of smooth muscle occurs. To evaluate the mechanisms of lung i...

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Veröffentlicht in:	Environmental research 1990-10, Vol.53 (1), p.16-28
1. Verfasser:	Rom, William N.
Format:	Artikel
Sprache:	eng
Schlagworte:	01 COAL, LIGNITE, AND PEAT 016000 - Coal, Lignite, & Peat- Health & Safety Adult AEROSOLS alpha 1-Antitrypsin - analysis ANIMAL CELLS ANIMAL TISSUES Biological and medical sciences BIOLOGICAL MATERIALS BLOOD BLOOD CELLS BODY BODY FLUIDS Bronchoalveolar Lavage Fluid - chemistry Bronchoalveolar Lavage Fluid - enzymology Bronchoalveolar Lavage Fluid - pathology Chemical and industrial products toxicology. Toxic occupational diseases COAL MINERS COAL MINING COLLOIDS CONNECTIVE TISSUE CELLS DISEASES DISPERSIONS EMPHYSEMA ENZYMES EPITHELIUM Female Humans HYDROLASES Inorganic dusts (pneumoconiosises) and organic dusts (byssinosis etc.) LAVAGE LEUKOCYTES Lung - diagnostic imaging Lung - enzymology Lung - pathology Lung - physiopathology LUNGS MACROPHAGES Macrophages - metabolism Male MATERIALS Medical sciences MINERS MINING NEUTROPHILS ORGANS Pancreatic Elastase - analysis PATHOGENESIS PEPTIDE HYDROLASES PERSONNEL PHAGOCYTES PNEUMOCONIOSES Pneumoconiosis - complications Pneumoconiosis - pathology Pulmonary Emphysema - etiology Pulmonary Emphysema - pathology Radionuclide Imaging RESIDUES RESPIRATORY SYSTEM RESPIRATORY SYSTEM DISEASES SERINE PROTEINASES SMOKES Smoking SOLS SOMATIC CELLS TISSUES TOBACCO SMOKES Toxicology
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