Neuromuscular Contacts Induce Nitric Oxide Signals in Skeletal Myotubes in vitro
It has previously been shown that skeletal myotubes express nitric oxide synthase (NOS) and produce and release NO signals. NOS is also part of agrin-induced acetylcholine receptor aggregations on myotubes. As nerve-muscle interactions underlie reciprocal signaling mechanisms, we hypothesized that N...
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Veröffentlicht in: | Neuro-Signals 2005-01, Vol.14 (3), p.85-95 |
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Sprache: | eng |
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