IL-32-dependent effects of IL-1β on endothelial cell functions

Increasing evidence demonstrates that interleukin (IL)-32 is a pro-inflammatory cytokine, inducing IL-1α, IL-1β, IL-6, tumor necrosis factor (TNF)-α, and chemokines via nuclear factor (NF)-κB, p38 mitogen-activated protein kinase (MAPK), and activating protein (AP)-1 activation. Here we report that...

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Veröffentlicht in:	Proceedings of the National Academy of Sciences - PNAS 2009-03, Vol.106 (10), p.3883-3888
Hauptverfasser:	Nold-Petry, Claudia A, Nold, Marcel F, Zepp, Jarod A, Kim, Soo-Hyun, Voelkel, Norbert F, Dinarello, Charles A
Format:	Artikel
Sprache:	eng
Schlagworte:	Atherosclerosis Biological Sciences Coagulation Cytokines Endothelial cells Endothelium Human umbilical vein endothelial cells Inflammation Messenger RNA Platelets Protein isoforms Proteins Q1 Small interfering RNA tumors
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