TNF-alpha inhibits isoproterenol-stimulated adenylyl cyclase activity in cultured airway smooth muscle cells

C. W. Emala, J. Kuhl, C. L. Hungerford and C. A. Hirshman Department of Anesthesiology, The Johns Hopkins Medical Institutions, Baltimore, Maryland 21205, USA. Inflammation, increased cytokine production, and decreased responsiveness of airway smooth muscle (ASM) to beta-adrenergic agonists are char...

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Veröffentlicht in:	American journal of physiology. Lung cellular and molecular physiology 1997-04, Vol.272 (4), p.644-L650
Hauptverfasser:	Emala, C. W, Kuhl, J, Hungerford, C. L, Hirshman, C. A
Format:	Artikel
Sprache:	eng
Schlagworte:	Adenosine Diphosphate Ribose - metabolism Adenylate Cyclase Toxin Adenylyl Cyclase Inhibitors Adenylyl Cyclases - metabolism Adrenergic beta-Agonists - pharmacology Animals Cells, Cultured Dogs GTP-Binding Proteins - metabolism Isoproterenol - pharmacology Muscle, Smooth - cytology Muscle, Smooth - drug effects Muscle, Smooth - enzymology Trachea - cytology Trachea - drug effects Trachea - enzymology Tumor Necrosis Factor-alpha - pharmacology Virulence Factors, Bordetella - pharmacology
Online-Zugang:	Volltext
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