Modulation of actin isoform expression before the transition from experimental compensated pressure-overload cardiac hypertrophy to decompensation

1 Department of Evolutionary and Functional Biology, Physiology Section, University of Parma, Italy; and 2 Departments of Pathology and Immunology and 3 Clinical Pathology, University of Geneva, Switzerland Submitted 2 October 2008 ; accepted in final form 21 February 2009 In a rat model of long-las...

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Veröffentlicht in:	American journal of physiology. Heart and circulatory physiology 2009-05, Vol.296 (5), p.H1625-H1632
Hauptverfasser:	Berni, Roberta, Savi, Monia, Bocchi, Leonardo, Delucchi, Francesca, Musso, Ezio, Chaponnier, Christine, Gabbiani, Giulio, Clement, Sophie, Stilli, Donatella
Format:	Artikel
Sprache:	eng
Schlagworte:	Actins - metabolism Adaptation, Physiological Animals Arterioles - metabolism Calcium Signaling Cardiomegaly - etiology Cardiomegaly - metabolism Cardiomegaly - physiopathology Cells Coronary Vessels - metabolism Disease Models, Animal Fibrosis Gene expression Heart Failure - etiology Heart Failure - metabolism Heart Failure - physiopathology Hemodynamics Hypertension - complications Hypertension - metabolism Hypertension - physiopathology Male Myocardial Contraction Myocardium - metabolism Myocardium - pathology Protein Isoforms Rats Rats, Wistar Rodents Studies Time Factors Transitions Ventricular Dysfunction, Left - etiology Ventricular Dysfunction, Left - metabolism Ventricular Dysfunction, Left - physiopathology Ventricular Function, Left
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