Cardiomyocytes from postinfarction failing rat hearts have improved ischemia tolerance

1 Clinical Chemistry Department and 2 Institute for Experimental Medical Research, Ullevål University Hospital, Oslo; 3 Center for Heart Failure Research, University of Oslo, Oslo; and 4 Department of Cardiology, Ullevål University Hospital, Oslo, Norway Submitted 31 July 2008 ; accepted in final fo...

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Veröffentlicht in:	American journal of physiology. Heart and circulatory physiology 2009-03, Vol.296 (3), p.H787-H795
Hauptverfasser:	Sharikabad, Mohammad Nouri, Aronsen, Jan Magnus, Haugen, Espen, Pedersen, Janne, Moller, Anne-Sophie W, Mork, Halvor Kjeang, Aass, Hans C. D, Sejersted, Ole M, Sjaastad, Ivar, Brors, Odd
Format:	Artikel
Sprache:	eng
Schlagworte:	Acetanilides - pharmacology Adenosine triphosphatase Adenosine Triphosphate - metabolism Animals Apoptosis Calcium Calcium - metabolism Cell culture Cell Death Cell Hypoxia Cells, Cultured Disease Models, Animal Enzymes Heart failure Heart Failure - etiology Heart Failure - metabolism Heart Failure - pathology L-Lactate Dehydrogenase - metabolism Male Myocardial Infarction - complications Myocardial Infarction - metabolism Myocardial Infarction - pathology Myocardial Reperfusion Injury - etiology Myocardial Reperfusion Injury - metabolism Myocardial Reperfusion Injury - prevention & control Myocytes, Cardiac - drug effects Myocytes, Cardiac - enzymology Myocytes, Cardiac - metabolism Myocytes, Cardiac - pathology Oxygen - metabolism Piperazines - pharmacology Potassium - metabolism Ranolazine Rats Rats, Wistar Rodents Rubidium Radioisotopes Sodium - metabolism Sodium Channel Blockers - pharmacology Sodium-Calcium Exchanger - metabolism Sodium-Potassium-Exchanging ATPase - metabolism Studies Time Factors
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