Ventricular-arterial coupling in a rat model of reduced arterial compliance provoked by hypervitaminosis D and nicotine

1 Laboratory of Hemodynamics and Cardiovascular Technology, Ecole Polytechnique Fédérale de Lausanne, Lausanne; 2 Department of Cardiovascular Surgery, Centre Hospitalière Universitaire Vardois, Lausanne; 4 Department of Cardiovascular Surgery, Inselspital, Bern; 3 Department of Cardiology, Centre H...

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Veröffentlicht in:	American journal of physiology. Heart and circulatory physiology 2006-10, Vol.291 (4), p.H1942-H1951
Hauptverfasser:	Jegger, David, da Silva, Rafaela, Jeanrenaud, Xavier, Nasratullah, Mohammad, Tevaearai, Hendrik, von Segesser, Ludwig K, Segers, Patrick, Gaillard, Virginie, Atkinson, Jeffrey, Lartaud, Isabelle, Stergiopulo, Nikolaos
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Calcium Cardiac Volume - drug effects Cardiac Volume - physiology Cardiovascular disease Cholecalciferol - pharmacology Compliance Coronary Vessels - drug effects Coronary Vessels - physiology Dose-Response Relationship, Drug Elasticity - drug effects Ganglionic Stimulants - pharmacology Heart Ventricles - drug effects Hemodynamics - drug effects Hemodynamics - physiology Hypertension Hypertension - physiopathology Male Models, Cardiovascular Nicotine Nicotine - pharmacology Rats Rats, Wistar Vascular Resistance - drug effects Vascular Resistance - physiology Ventricular Function Vitamin D
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