Bcl-2 Overexpression Leads to Increases in Suppressor of Cytokine Signaling-3 Expression in B Cells and De novo Follicular Lymphoma11NIH grants CA78254 (G.J. Vanasse) and 5U24DK058813-02 (K.Y. Yeung), American Society of Hematology fellow scholar grant (G.J. Vanasse), and NIH research grant CA-16359 from the National Cancer Institute.Notes: G.J. Vanasse is a past American Society of Hematology fellow scholar and member of the Yale Cancer Center

The t(14;18)(q32;q21), resulting in deregulated expression of B-cell-leukemia/lymphoma-2 (Bcl-2), represents the genetic hallmark in human follicular lymphomas. Substantial evidence supports the hypothesis that the t(14;18) and Bcl-2 overexpression are necessary but not solely responsible for neopla...

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Veröffentlicht in:	Molecular cancer research 2004-11, Vol.2 (11), p.620
Hauptverfasser:	Gary J. Vanasse, Robert K. Winn, Sofya Rodov, Arthur W. Zieske, John T. Li, Joan C. Tupper, Jingjing Tang, Elaine W. Raines, Mette A. Peters, Ka Yee Yeung, John M. Harlan
Format:	Artikel
Sprache:	eng
Schlagworte:	B cells Bcl-2 follicular lymphoma microarray SOCS3 gene
Online-Zugang:	Volltext
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