Genetic differentiation of Poly I:C from B:9-23 peptide induced experimental autoimmune diabetes

Type 1 diabetes is an immune-mediated disease, in which T cells of the adaptive immune system mediate beta cell destruction. Recently the innate immune system has been linked to etiopathogenesis of several autoimmune diseases including type 1 diabetes, as innate effector cells (e.g. dendritic cells,...

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Veröffentlicht in:	Journal of autoimmunity 2004-06, Vol.22 (4), p.307-313
Hauptverfasser:	Paronen, Johanna, Liu, Edwin, Moriyama, Hiroaki, Devendra, Devasenan, Ide, Akane, Taylor, Robert, Yu, Liping, Miao, Dongmei, Melanitou, Evie, Eisenbarth, George S.
Format:	Artikel
Sprache:	eng
Schlagworte:	Animal genetics Animals Autoantigens Autoantigens - administration & dosage B7-1 Antigen B7-1 Antigen - genetics B:9-23 peptide Biological and medical sciences Cellular Biology Diabetes Mellitus, Type 1 Diabetes Mellitus, Type 1 - etiology Diabetes Mellitus, Type 1 - genetics Diabetes Mellitus, Type 1 - immunology Diabetes Mellitus, Type 1 - pathology Endocrinology and metabolism Experimental autoimmune diabetes General aspects Genetics Human genetics Human health and pathology Immunity, Innate Immunopathology Infectious diseases Insulin Insulin - administration & dosage Insulin - immunology Islets of Langerhans Islets of Langerhans - immunology Islets of Langerhans - pathology Life Sciences Medical sciences Mice Mice, Inbred BALB C Mice, Inbred C57BL Mice, Transgenic Peptide Fragments Peptide Fragments - administration & dosage Peptide Fragments - immunology Poly I-C Poly I-C - administration & dosage Poly I-C - immunology Rats Species Specificity Subcellular Processes
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