Haploinsufficiency of the IKZF1 (IKAROS) tumor suppressor gene cooperates with BCR-ABL in a transgenic model of acute lymphoblastic leukemia

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Veröffentlicht in:	Leukemia 2010-06, Vol.24 (6), p.1200-1204
Hauptverfasser:	Virely, C, Moulin, S, Cobaleda, C, Lasgi, C, Alberdi, A, Soulier, J, Sigaux, F, Chan, S, Kastner, P, Ghysdael, J
Format:	Artikel
Sprache:	eng
Schlagworte:	631/67/581 692/420/755 692/699/67/1990/283/2125 Acute lymphoblastic leukemia Acute lymphocytic leukemia Animals B cells BCR-ABL protein Biological and medical sciences Cancer Cancer Research Cell receptors Chromosome aberrations Critical Care Medicine Development and progression Female Flow Cytometry Fusion protein Fusion Proteins, bcr-abl - physiology Genes, Tumor Suppressor - physiology Genetic aspects Haploinsufficiency Hematologic and hematopoietic diseases Hematology Humans Ikaros protein Ikaros Transcription Factor - physiology Intensive Internal Medicine letter-to-the-editor Leukemia Leukemias. Malignant lymphomas. Malignant reticulosis. Myelofibrosis Life Sciences Lymphatic leukemia Male Medical genetics Medical sciences Medicine Medicine & Public Health Mice Mice, Inbred C57BL Mice, Transgenic Oncology Physiological aspects Precursor Cell Lymphoblastic Leukemia-Lymphoma - etiology Precursor Cell Lymphoblastic Leukemia-Lymphoma - pathology Survival Rate Transcription factors Tumor suppressor genes Tumors
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