High salt diet‐induced proximal tubular phenotypic changes and sodium‐glucose cotransporter‐2 expression are coordinated by cold shock Y‐box binding protein‐1

High salt diet (HSD) is a hallmark of blood pressure elevations, weight gain and diabetes onset in the metabolic syndrome. In kidney, compensatory mechanisms are activated to balance salt turnover and maintain homeostasis. Data on the long‐term effects of HSD with respect to tubular cell functions a...

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Veröffentlicht in:	The FASEB journal 2021-10, Vol.35 (10), p.e21912-n/a
Hauptverfasser:	Bernhardt, Anja, Häberer, Saskia, Xu, JingJing, Damerau, Hannah, Steffen, Johannes, Reichardt, Charlotte, Wolters, Katharina, Steffen, Hannes, Isermann, Berend, Borucki, Katrin, Artelt, Nadine, Endlich, Nicole, Kozyraki, Renata, Brandt, Sabine, Lindquist, Jonathan A., Mertens, Peter R.
Format:	Artikel
Sprache:	eng
Schlagworte:	cold shock protein Endocrinology and metabolism high salt diet Human health and pathology Life Sciences sodium glucose transporter tubular damage
Online-Zugang:	Volltext
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