Decrease in calbindin content significantly alters LTP but not NMDA receptor and calcium channel properties

The contribution of the cytosolic calcium binding protein calbindin D 28K (CaBP) to the synaptic plasticity was investigated in hippocampal CA1 area of wild-type and antisense transgenic CaBP-deficient mice. We showed that long-term potentiation (LTP) induced by tetanic stimulation in CaBP-deficient...

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Veröffentlicht in:	Neuropharmacology 2002-03, Vol.42 (4), p.444-458
Hauptverfasser:	Jouvenceau, A, Potier, B, Poindessous-Jazat, F, Dutar, P, Slama, A, Epelbaum, J, Billard, J.-M
Format:	Artikel
Sprache:	eng
Schlagworte:	Aminoacid receptors (glycine, glutamate, gaba) Animals Binding Sites - genetics Biological and medical sciences Calbindin Calbindins Calcium binding protein Calcium channels Calcium Channels - genetics Calcium Channels - metabolism Calcium Channels - physiology Cell receptors Cell structures and functions Central nervous system Central neurotransmission. Neuromudulation. Pathways and receptors Fundamental and applied biological sciences. Psychology Glutamate Glycine Hippocampus Life Sciences Long-term potentiation Long-Term Potentiation - genetics Male Mice Mice, Transgenic Molecular and cellular biology Neuronal Plasticity - genetics Neurons and Cognition NMDA receptor Receptors, N-Methyl-D-Aspartate - genetics Receptors, N-Methyl-D-Aspartate - metabolism Receptors, N-Methyl-D-Aspartate - physiology S100 Calcium Binding Protein G - genetics S100 Calcium Binding Protein G - metabolism S100 Calcium Binding Protein G - physiology Vertebrates: nervous system and sense organs
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