The N-Myc-responsive lncRNA MILIP promotes DNA doublestrand break repair through non-homologous end joining
The protooncoprotein N-Myc, which is overexpressed in approximately 25% of neuroblastomas as the consequence of MYCN gene amplification, has long been postulated to regulate DNA double-strand break (DSB) repair in neuroblastoma cells, but experimental evidence of this function is presently scant. He...
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Veröffentlicht in: | Nevada RNformation 2022, p.1 |
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Hauptverfasser: | , , , , , , , , , , , , , , , , , , , , , , |
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Sprache: | eng |
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