TLR9-induced interferon [beta] is associated with protection from gammaherpesvirus-induced exacerbation of lung fibrosis

Background We have shown previously that murine gammaherpesvirus 68 ([gamma]HV68) infection exacerbates established pulmonary fibrosis. Because Toll-like receptor (TLR)-9 may be important in controlling the immune response to [gamma]HV68 infection, we examined how TLR-9 signaling effects exacerbatio...

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Veröffentlicht in:Fibrogenesis & tissue repair 2011-08, Vol.4, p.18
Hauptverfasser: Luckhardt, Tracy R, Coomes, Stephanie M, Trujillo, Glenda, Stoolman, Joshua S, Vannella, Kevin M, Bhan, Urvashi, Wilke, Carol A, Moore, Thomas A, Toews, Galen B, Hogaboam, Cory, Moore, Bethany B
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Sprache:eng
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