TLR9-induced interferon [beta] is associated with protection from gammaherpesvirus-induced exacerbation of lung fibrosis
Background We have shown previously that murine gammaherpesvirus 68 ([gamma]HV68) infection exacerbates established pulmonary fibrosis. Because Toll-like receptor (TLR)-9 may be important in controlling the immune response to [gamma]HV68 infection, we examined how TLR-9 signaling effects exacerbatio...
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Veröffentlicht in: | Fibrogenesis & tissue repair 2011-08, Vol.4, p.18 |
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