Defective Insulin-Induced GLUT4 Translocation in Skeletal Muscle of High Fat–Fed Rats Is Associated With Alterations in Both Akt/Protein Kinase B and Atypical Protein Kinase C (ζ/λ) Activities

Defective Insulin-Induced GLUT4 Translocation in Skeletal Muscle of High Fat–Fed Rats Is Associated With Alterations in Both Akt/Protein Kinase B and Atypical Protein Kinase C (ζ/λ) Activities Frédéric Tremblay 1 2 , Charles Lavigne 2 3 , Hélène Jacques 2 3 and André Marette 1 2 1 Department of Phys...

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Veröffentlicht in:	Diabetes (New York, N.Y.) N.Y.), 2001-08, Vol.50 (8), p.1901-1910
Hauptverfasser:	TREMBLAY, Frédéric, LAVIGNE, Charles, JACQUES, Hélène, MARETTE, André
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Biological and medical sciences Blood Glucose - metabolism Cell Membrane - metabolism Diabetes Dietary Fats - pharmacology Evaluation Fat Fats Feeding. Feeding behavior Fundamental and applied biological sciences. Psychology Glucose Glucose Transporter Type 4 Insulin Insulin - blood Insulin - pharmacology Insulin Receptor Substrate Proteins Insulin resistance Insulin Resistance - physiology Isoenzymes Kinases Kinetics Laboratory rats Male Microtubules - metabolism Monosaccharide Transport Proteins - metabolism Muscle Proteins Muscle, Skeletal - drug effects Muscle, Skeletal - metabolism Musculoskeletal system Phosphatidylinositol 3-Kinases - metabolism Phosphoproteins - metabolism Phosphorylation Plasma Protein Kinase C - metabolism Protein kinases Protein Transport - drug effects Protein-Serine-Threonine Kinases Proteins Proto-Oncogene Proteins - metabolism Proto-Oncogene Proteins c-akt Rats Rats as laboratory animals Rats, Wistar Signal transduction Striated muscle Tumor necrosis factor-TNF Vertebrates: anatomy and physiology, studies on body, several organs or systems
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