Hyperketonemia Increases Tumor Necrosis Factor-α Secretion in Cultured U937 Monocytes and Type 1 Diabetic Patients and Is Apparently Mediated by Oxidative Stress and cAMP Deficiency

Hyperketonemia Increases Tumor Necrosis Factor-α Secretion in Cultured U937 Monocytes and Type 1 Diabetic Patients and Is Apparently Mediated by Oxidative Stress and cAMP Deficiency Sushil K. Jain , Krishnaswamy Kannan , Gideon Lim , Robert McVie and Joseph A. Bocchini, Jr. From the Department of Pe...

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Veröffentlicht in:	Diabetes (New York, N.Y.) N.Y.), 2002-07, Vol.51 (7), p.2287-2293
Hauptverfasser:	JAIN, Sushil K, KANNAN, Krishnaswamy, LIM, Gideon, MCVIE, Robert, BOCCHINI, Joseph A
Format:	Artikel
Sprache:	eng
Schlagworte:	3-Hydroxybutyric Acid - pharmacology Acetoacetates - pharmacology Acetylcysteine - pharmacology Antioxidants - pharmacology Apoptosis Associated diseases and complications Atherosclerosis Biological and medical sciences Cell culture Complications and side effects Cyclic AMP - blood Cyclic AMP - physiology Development and progression Diabetes Diabetes mellitus Diabetes Mellitus, Type 1 - blood Diabetes Mellitus, Type 1 - physiopathology Diabetes. Impaired glucose tolerance Endocrine pancreas. Apud cells (diseases) Endocrinopathies Glucose Humans Hyperglycemia Hypotheses Ketone Bodies - blood Ketosis Kinases Medical sciences Mitogen-Activated Protein Kinases - metabolism Monocytes - drug effects Monocytes - physiology Oxidative stress Oxidative Stress - physiology Peripheral vascular diseases Physiological aspects Protein Kinase C - blood Proteins Reference Values Tetradecanoylphorbol Acetate - pharmacology Transcription factors Tumor necrosis factor Tumor Necrosis Factor-alpha - metabolism Tumor necrosis factor-TNF Tumour necrosis factor U937 Cells
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