Additional file 5 of Loss of miR-26b-5p promotes gastric cancer progression via miR-26b-5p-PDE4B/CDK8-STAT3 feedback loop

Additional file 5 of Loss of miR-26b-5p promotes gastric cancer progression via miR-26b-5p-PDE4B/CDK8-STAT3 feedback loop

Additional file 5: Figure S5. (A-D) EdU, colony formation, and CCK8 analysis of MGC803 cells transfected with siPDE4B or siCDK8. (E–H) colony formation, EdU and CCK8 assays were used to assess the proliferation capacity of MGC803 cells transfected with Ctrl inhibitor + si-NC, 26b-5p inhibitor + si-N...

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Bibliographische Detailangaben
Hauptverfasser: Xu, Tingting, Xie, Mengyan, Jing, Xinming, Jiang, Huning, Wu, Xi, Wang, Xinzhu, Shu, Yongqian
Format: Bild
Sprache:eng
Schlagworte:
Biochemistry
Biological Sciences not elsewhere classified
Biophysics
Cancer
Cell Biology
Chemical Sciences not elsewhere classified
Computational Biology
FOS: Biological sciences
FOS: Clinical medicine
FOS: Health sciences
Genetics
Immunology
Infectious Diseases
Marine Biology
Medicine
Molecular Biology
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Zusammenfassung:Additional file 5: Figure S5. (A-D) EdU, colony formation, and CCK8 analysis of MGC803 cells transfected with siPDE4B or siCDK8. (E–H) colony formation, EdU and CCK8 assays were used to assess the proliferation capacity of MGC803 cells transfected with Ctrl inhibitor + si-NC, 26b-5p inhibitor + si-NC, 26b-5p inhibitor + siCDK8, or 26b-5p inhibitor + siPDE4B. Quantitative data are shown as the mean ± SD of three independent experiments. *P 
DOI:10.6084/m9.figshare.22613893