MOESM5 of Loss of amyloid precursor protein exacerbates early inflammation in Niemann-Pick disease type C

Additional file 5: Figure S5. Loss of APP function results in the exacerbation of DEGs functionally related to antiviral response in Npc1-/-/App-/- mouse cerebella. All differentially expressed genes (DEGs) are localized to their sub-cellular location. All plotted DEGs meet the significance cutoff o...

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Hauptverfasser: Shin, Samuel, Shin, Alexandra, Mayagoitia, Karina, Siebold, Lorraine, Rubini, Marsilio, Wilson, Christopher, Bellinger, Denise, Soriano, Salvador
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creator Shin, Samuel
Shin, Alexandra
Mayagoitia, Karina
Siebold, Lorraine
Rubini, Marsilio
Wilson, Christopher
Bellinger, Denise
Soriano, Salvador
description Additional file 5: Figure S5. Loss of APP function results in the exacerbation of DEGs functionally related to antiviral response in Npc1-/-/App-/- mouse cerebella. All differentially expressed genes (DEGs) are localized to their sub-cellular location. All plotted DEGs meet the significance cutoff of fold-change (absolute FC > 1.5) and p-value (p < 0.05). *Duplicate identifiers used for the same gene. A detailed key for IPA molecular shape, color, and interaction is provided in Fig. 2.
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identifier DOI: 10.6084/m9.figshare.11393514
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subjects Biochemistry
Biophysics
Cell Biology
Developmental Biology
FOS: Biological sciences
FOS: Health sciences
Genetics
Infectious Diseases
Medicine
Mental Health
Molecular Biology
Neuroscience
Physiology
title MOESM5 of Loss of amyloid precursor protein exacerbates early inflammation in Niemann-Pick disease type C
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