Recruitment of RBM6 to DNA double-strand breaks fosters homologous recombination repair
DNA double-strand breaks (DSBs) are highly toxic lesions that threaten genome integrity and cell survival. To avoid harmful repercussions of DSBs, a wide variety of DNA repair factors are recruited to execute DSB repair by either non-homologous end joining (NHEJ), homologous recombination (HR), or a...
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