Signal transduction of Helicobacter pylori during interaction with host cell protein receptors of epithelial and immune cells

Helicobacter pylori infections can induce pathologies ranging from chronic gastritis, peptic ulceration to gastric cancer. Bacterial isolates harbor numerous well-known adhesins, vacuolating cytotoxin VacA, protease HtrA, urease, peptidoglycan, and type IV secretion systems (T4SS). It appears that H...

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Veröffentlicht in:	Gut microbes 2013-11, Vol.4 (6), p.454-474
Hauptverfasser:	Pachathundikandi, Suneesh Kumar, Tegtmeyer, Nicole, Backert, Steffen
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Bacterial Proteins - metabolism Bacterial Secretion Systems c-Met Disease Models, Animal E-cadherin EGF receptor Epithelial Cells - metabolism Helicobacter Infections - metabolism Helicobacter pylori - metabolism Host-Pathogen Interactions Humans Immunity, Innate integrins molecular pathogenesis Receptors, Cell Surface - metabolism Review Signal Transduction virulence
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