C-Type Lectin SIGN-R1 Has a Role in Experimental Colitis and Responsiveness to Lipopolysaccharide

Pathogen recognition receptors (PRRs) function to maintain the balance between controlled responses to pathogens and uncontrolled innate immune activation leading to inflammation. In the context of commensal bacteria and the etiology of inflammatory bowel disease, although a role for the TLRs is kno...

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Veröffentlicht in:	The Journal of immunology (1950) 2010-03, Vol.184 (5), p.2627-2637
Hauptverfasser:	Saunders, Sean P, Barlow, Jillian L, Walsh, Caitriona M, Bellsoi, Agustin, Smith, Philip, McKenzie, Andrew N. J, Fallon, Padraic G
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Cell Adhesion Molecules - genetics Cell Adhesion Molecules - metabolism Cell Adhesion Molecules - physiology Colitis - chemically induced Colitis - genetics Colitis - physiopathology Colon - drug effects Colon - metabolism Colon - pathology Dextran Sulfate Female Flow Cytometry Fluorescent Antibody Technique Genetic Predisposition to Disease Interleukin-18 - metabolism Interleukin-1beta - metabolism Interleukin-6 - metabolism Lectins, C-Type - genetics Lectins, C-Type - metabolism Lectins, C-Type - physiology Lipopolysaccharides - toxicity Macrophages, Peritoneal - drug effects Macrophages, Peritoneal - metabolism Male Mice Mice, Inbred C57BL Mice, Knockout Myeloid Differentiation Factor 88 - deficiency Myeloid Differentiation Factor 88 - genetics Receptors, Cell Surface - genetics Receptors, Cell Surface - metabolism Receptors, Cell Surface - physiology Shock - chemically induced Shock - genetics Shock - physiopathology Toll-Like Receptor 2 - deficiency Toll-Like Receptor 2 - genetics Toll-Like Receptor 4 - deficiency Toll-Like Receptor 4 - genetics Tumor Necrosis Factor-alpha - metabolism
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