Platelet-rich plasma induces increased expression of G1 cell cycle regulators, type I collagen, and matrix metalloproteinase-1 in human skin fibroblasts
Platelet-rich plasma (PRP) is derived from fresh whole blood, which contains a high concentration of platelets. Recently, PRP has been used for skin wound healing and rejuvenation. However, the molecular mechanisms underlying PRP-inducing wound healing processes are still largely unknown. The aim of...
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Veröffentlicht in: | International journal of molecular medicine 2012-01, Vol.29 (1), p.32-36 |
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Zusammenfassung: | Platelet-rich plasma (PRP) is derived from fresh whole blood, which contains
a high concentration of platelets. Recently, PRP has been used for skin wound
healing and rejuvenation. However, the molecular mechanisms underlying PRP-inducing
wound healing processes are still largely unknown. The aim of this study is to
evaluate the effect of PRP on the expression of G1 cell cycle regulatory proteins,
type I collagen, matrix metalloproteinase-1 (MMP-1), and MMP-2 in human skin fibroblasts
(HSF). We performed a cell proliferation and a migration assay, immunoblotting,
and a chloramphenicol acetyltransferase (CAT) assay in PRP-treated human skin
fibroblasts. PRP treatment induced increased rates of cell proliferation and cell
migration. Expression of cyclin A protein was increased by a low concentration
(0.5%) of PRP-treated HSF. In addition, expression of Rb, cyclin E, and cyclin-dependent
kinase 4 proteins was increased by a high concentration (5%) of PRP-treated HSF.
High concentration of PRP induced an up-regulation of type I collagen, MMP-1,
and MMP-2 expression in HSF. Taken together, PRP treatment induced an increase
in expression of G1 cell cycle regulators, type I collagen and MMP-1, thereby
accelerating the wound healing process. |
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ISSN: | 1107-3756 1791-244X |
DOI: | 10.3892/ijmm.2011.803 |