Nitrous oxide does not hinder the repair of halothane-induced hepatic injury in the rat

Nitrous oxide administration may limit DNA synthesis by inactivating methionine synthetase, and may thus hamper the repair of an injured organ such as the liver. To test this possibility, we pretreated rats with phenobarbital and exposed them to 0.3 MAC halothane in 9% oxygen for 46 min, followed im...

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Veröffentlicht in:	Anesthesia and analgesia 1985-05, Vol.64 (5), p.465-467
Hauptverfasser:	FASSOULAKI, A, EGER, E. I. II, JOHNSON, B. H, FERRELL, L. D, SMUCKLER, E. A, CAHALAN, M. K, EGER, R. R, HARPER, M. H
Format:	Artikel
Sprache:	eng
Schlagworte:	5-Methyltetrahydrofolate-Homocysteine S-Methyltransferase - antagonists & inhibitors Anesthetics. Neuromuscular blocking agents Animals Biological and medical sciences Bretylium Compounds - therapeutic use Chemical and Drug Induced Liver Injury DNA - biosynthesis Halothane - toxicity Isoflurane - toxicity Liver Diseases - pathology Liver Diseases - physiopathology Liver Regeneration - drug effects Male Medical sciences Neuropharmacology Nitrous Oxide - toxicity Pharmacology. Drug treatments Phenobarbital - pharmacology Rats Rats, Inbred Strains
Online-Zugang:	Volltext
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