Curcumin Requires Tumor Necrosis Factor α Signaling to Alleviate Cognitive Impairment Elicited by Lipopolysaccharide

A decline in cognitive ability is a typical feature of the normal aging process, and of neurodegenerative disorders such as Alzheimer's, Parkinson's and Huntington's diseases. Although their etiologies differ, all of these disorders involve local activation of innate immune pathways a...

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Veröffentlicht in:	Neuro-Signals 2013, Vol.21 (1-2), p.75-88
Hauptverfasser:	Kawamoto, E.M., Scavone, C., Mattson, M.P., Camandola, S.
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Cells, Cultured Cognition Cognition Disorders - chemically induced Cognition Disorders - drug therapy Cognition Disorders - metabolism Curcumin Curcumin - pharmacology Curcumin - therapeutic use Inflammation Lipopolysaccharide Lipopolysaccharides - toxicity Male Maze Learning - drug effects Maze Learning - physiology Mice Mice, Inbred C57BL Mice, Knockout Original Paper Receptors, Tumor Necrosis Factor, Type II - metabolism Signal Transduction - drug effects Signal Transduction - physiology Tumor necrosis factor α Tumor Necrosis Factor-alpha - metabolism
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