Mycobacterium tuberculosis Lipoproteins Directly Regulate Human Memory CD4⁺ T Cell Activation via Toll-Like Receptors 1 and 2

The success of Mycobacterium tuberculosis as a pathogen relies on its ability to regulate the host immune response. M. tuberculosis can manipulate adaptive T cell responses indirectly by modulating antigen-presenting cell (APC) function or by directly interacting with T cells. Little is known about...

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Veröffentlicht in:	Infection and Immunity 2011-02, Vol.79 (2), p.663-673
Hauptverfasser:	Lancioni, Christina L, Li, Qing, Thomas, Jeremy J, Ding, XueDong, Thiel, Bonnie, Drage, Michael G, Pecora, Nicole D, Ziady, Assem G, Shank, Samuel, Harding, Clifford V, Boom, W. Henry, Rojas, Roxana E
Format:	Artikel
Sprache:	eng
Schlagworte:	Acylation adaptive immunity Adult antibodies Bacteriology Biological and medical sciences CD4-positive T-lymphocytes CD4-Positive T-Lymphocytes - immunology cell proliferation Cells, Cultured Fundamental and applied biological sciences. Psychology Gene Expression Regulation Host Response and Inflammation Humans immune response Immunologic Memory - physiology lipoproteins Lipoproteins - genetics Lipoproteins - immunology Lipoproteins - metabolism Lymphocyte Activation - physiology Microbiology Middle Aged Miscellaneous Mycobacterium tuberculosis Mycobacterium tuberculosis - immunology Mycobacterium tuberculosis - metabolism pathogens receptors secretion Toll-Like Receptor 1 - genetics Toll-Like Receptor 1 - metabolism Toll-Like Receptor 2 - genetics Toll-Like Receptor 2 - metabolism Young Adult
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