Aminothiols protect endothelial cell proliferation against inhibition by lipopolysaccharide

Lipopolysaccharide (LPS) is a primary agent of sepsis that damages the vascular endothelium. Endothelial cell proliferation is key to the repair of damaged endothelium, and drugs that counteract the antiproliferative impact of LPS on endothelial cells should be beneficial. Because LPS exerts much of...

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Veröffentlicht in:	Shock (Augusta, Ga.) Ga.), 1998-12, Vol.10 (6), p.423-429
Hauptverfasser:	DRAB-WEISS, E. A, HANSRA, I. K, BLAZEK, E. R, RUBIN, D. B
Format:	Artikel
Sprache:	eng
Schlagworte:	Anesthesia. Intensive care medicine. Transfusions. Cell therapy and gene therapy Animals Apoptosis - drug effects Biological and medical sciences Cattle Cell Division - drug effects DNA - biosynthesis DNA Damage Emergency and intensive care: infection, septic shock Endothelium, Vascular - cytology Endothelium, Vascular - drug effects Endothelium, Vascular - injuries In Vitro Techniques Intensive care medicine Lipopolysaccharides - toxicity Male Medical sciences Mercaptoethylamines - pharmacology Sepsis - drug therapy Sepsis - pathology
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