Carbon dot targeting to nitrogen signaling molecules for inhibiting neuronal death

Free radical-induced oxidative damage and nitrosative stress have been identified as key factors in neuroinflammation responses after traumatic brain injury (TBI), with which reactive oxygen and nitrogen species (RONS), especially nitrogen signaling molecules, are strongly associated. Here, we prepa...

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Veröffentlicht in:	Journal of materials chemistry. B, Materials for biology and medicine Materials for biology and medicine, 2020-03, Vol.8 (11), p.2321-233
Hauptverfasser:	Ouyang, Lufei, Mu, Xiaoyu, Wang, Junying, Li, Qifeng, Gao, Yalong, Liu, Haile, Sun, Si, Ren, Qinjuan, Yan, Ruijuan, Wang, Jingya, Liu, Qiang, Sun, Yuanming, Liu, Changlong, He, Hua, Long, Wei, Zhang, Xiao-Dong
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Antioxidants - chemistry Antioxidants - pharmacology Biological Transport Blood-Brain Barrier - metabolism Brain Injuries, Traumatic - drug therapy Carbon - chemistry Carbon dots Cell Death - drug effects Cysteine - chemistry Disease Models, Animal Experiments Free radicals Free Radicals - chemistry Free Radicals - metabolism Head injuries Humans In vitro methods and tests Inflammation Lipid peroxidation Lipid Peroxidation - drug effects Lipids Lysine - chemistry Mice Mice, Inbred C57BL Morris Water Maze Test - drug effects Neurons - cytology Nitrogen Oxidative Stress - drug effects Oxygen Peroxidation Quantum Dots - chemistry Quantum Dots - metabolism Reactive nitrogen species Reactive Nitrogen Species - chemistry Reactive Nitrogen Species - metabolism Reactive oxygen species Reactive Oxygen Species - chemistry Reactive Oxygen Species - metabolism Scavenging Signaling Superoxide dismutase Superoxide Dismutase - metabolism Tissue Distribution Traumatic brain injury
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