Reactive oxygen species mediate crosstalk between NF-κB and JNK

The activation of NF- κ B inhibits apoptosis via a mechanism involving upregulation of various antiapoptotic genes, such as cellular FLICE-inhibitory protein (c-FLIP), Bcl-x L , A1/Bfl-1 , and X chromosome-liked inhibitor of apoptosis (XIAP). In contrast, the activation of c-Jun N-terminal kinase (J...

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Veröffentlicht in:	Cell death and differentiation 2006-05, Vol.13 (5), p.730-737
Hauptverfasser:	Nakano, H, Nakajima, A, Sakon-Komazawa, S, Piao, J-H, Xue, X, Okumura, K
Format:	Artikel
Sprache:	eng
Schlagworte:	Apoptosis Biochemistry Biomedical and Life Sciences Cell Biology Cell Cycle Analysis Life Sciences review Stem Cells
Online-Zugang:	Volltext
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