Treatment of experimentally induced transient cerebral ischemia with low energy laser inhibits nitric oxide synthase activity and up-regulates the expression of transforming growth factor-beta 1

Background and Objectives Nitric oxide (NO) has been shown to be neurotoxic while transforming growth factor‐beta 1 (TGF‐β1) is neuroprotective in the stroke model. The present study investigates the effects of low energy laser on nitric oxide synthase (NOS) and TGF‐β1 activities after cerebral isch...

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Veröffentlicht in:	Lasers in surgery and medicine 2002-01, Vol.31 (4), p.283-288
Hauptverfasser:	Leung, Mason C.P., Lo, Samuel C.L., Siu, Flora K.W., So, K.-F.
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Biological and medical sciences Disease Models, Animal Diseases of the nervous system Ischemic Attack, Transient - physiopathology Ischemic Attack, Transient - radiotherapy laser Low-Level Light Therapy Male Medical sciences Middle Cerebral Artery - physiopathology Middle Cerebral Artery - radiation effects nitric oxide Nitric Oxide Synthase - physiology Nitric Oxide Synthase - radiation effects Radiotherapy. Instrumental treatment. Physiotherapy. Reeducation. Rehabilitation, orthophony, crenotherapy. Diet therapy and various other treatments (general aspects) Rats Rats, Sprague-Dawley Reperfusion Injury - physiopathology Reperfusion Injury - radiotherapy stroke Time Factors transforming growth factor Transforming Growth Factor beta - analysis Transforming Growth Factor beta - radiation effects Transforming Growth Factor beta1 Up-Regulation - physiology Up-Regulation - radiation effects
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