Energy substrate metabolism, mitochondrial structure and oxidative stress after cardiac ischemia-reperfusion in mice lacking UCP3

Myocardial ischemia-reperfusion (IR) injury may result in cardiomyocyte dysfunction. Mitochondria play a critical role in cardiomyocyte recovery after IR injury. The mitochondrial uncoupling protein 3 (UCP3) has been proposed to reduce mitochondrial reactive oxygen species (ROS) production and to fa...

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Veröffentlicht in:	Free radical biology & medicine 2023-08, Vol.205, p.244-261
Hauptverfasser:	Sánchez-Pérez, Patricia, Mata, Ana, Torp, May-Kristin, López-Bernardo, Elia, Heiestad, Christina M., Aronsen, Jan Magnus, Molina-Iracheta, Antonio, Jiménez-Borreguero, Luis J., García-Roves, Pablo, Costa, Ana S.H., Frezza, Christian, Murphy, Michael P., Stenslokken, Kåre-Olav, Cadenas, Susana
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Coronary Artery Disease - metabolism Energy Metabolism Fatty Acids - metabolism Infarction - complications Infarction - metabolism Ischemia - metabolism Ischemia-reperfusion injury Mice Mitochondria - metabolism Mitochondrial respiration Mitochondrial structure Myocardial Ischemia - metabolism Myocardial Reperfusion Injury - genetics Myocardial Reperfusion Injury - metabolism Myocytes, Cardiac - metabolism Oxidative Stress Reperfusion Superoxides - metabolism UCP3 (uncoupling protein 3)
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