Associations between complement pathways activity, mannose-binding lectin, and odds of unprovoked venous thromboembolism

Deep vein thrombosis (DVT) originates in the valvular sinuses of large veins in a local milieu characterized by stasis and severe hypoxia. This may induce complement- and coagulation activation, which potentially increases the risk of venous thromboembolism (VTE). The aim of the present study was to...

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Veröffentlicht in:	Thrombosis research 2018-09, Vol.169, p.50-56
Hauptverfasser:	Høiland, Ina Isabella, Liang, Robin Amanda, Hindberg, Kristian, Latysheva, Nadezhda, Brekke, Ole-Lars, Mollnes, Tom Eirik, Hansen, John-Bjarne
Format:	Artikel
Sprache:	eng
Schlagworte:	Adult Aged Blood Coagulation Case-Control Studies Clinical medical disciplines: 750 Complement Complement Activation Female Hematologi: 775 Hematology: 775 Humans Klinisk medisinske fag: 750 Male Mannose-Binding Lectin - blood Mannose-Binding Lectin - deficiency Mannose-Binding Lectin - immunology Medical disciplines: 700 Medisinske Fag: 700 Metabolism, Inborn Errors - blood Metabolism, Inborn Errors - complications Metabolism, Inborn Errors - immunology Middle Aged Thrombin - immunology Thrombin generation Thromboplastin - immunology Tissue factor VDP Venous Thromboembolism - blood Venous Thromboembolism - etiology Venous Thromboembolism - immunology Venous thrombosis
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