THE INFLUENCE OF STEATOSIS AND CONJUGATE FACTORS OF RESPONSE TO ANTIVIRAL TREATMENT IN CRONIC HEPATITIS C
HEPATITIS C VIRUS (HCV) IS A MAJOR CAUSE OF CHRONIC LIVER DISEASE WORLDWIDE. AN INCREASED PREVALENCE OF STEATOSIS IN PATIENTS WITH HCV IS WELL ESTABLISHED. MOSTSTUDIES HAVE REPORTED APPROXIMATELY 50% PREVALENCE OF STEATOSIS AMONG PATIENTSUNDERGOING A LIVER BIOPSY BECAUSE OF HCV. IN COMPARISON, AMONG...
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| Veröffentlicht in: | Research and science today 2016-03, Vol.11 (1), p.147-155 |
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| description | HEPATITIS C VIRUS (HCV) IS A MAJOR CAUSE OF CHRONIC LIVER DISEASE WORLDWIDE. AN INCREASED PREVALENCE OF STEATOSIS IN PATIENTS WITH HCV IS WELL ESTABLISHED. MOSTSTUDIES HAVE REPORTED APPROXIMATELY 50% PREVALENCE OF STEATOSIS AMONG PATIENTSUNDERGOING A LIVER BIOPSY BECAUSE OF HCV. IN COMPARISON, AMONG PATIENTS WITH AUTOIMMUNE HEPATITIS AND HEPATITIS B (HBV), STEATOSIS IS NOT COMMONLY OBSERVED. HEPATITIS C VIRUS (HCV) INFECTION IS AN IMPORTANT RISK FACTOR FOR INSULIN RESISTANCE(IR). THE LATTER IS THE PATHOGENIC FOUNDATION UNDERLYING METABOLIC SYNDROME,STEATOSIS AND CIRRHOSIS, AND POSSIBLY HEPATOCELLULAR CARCINOMA (HCC). WHEREAS THEOVERALL PREVALENCE OF IR IS 10%-25% OF THE POPULATION, THE PREVALENCE IR IN HCVINFECTION REACHES FIGURES RANGING BETWEEN 30% TO 70%. OBESITY AND/OR STEATOSIS INPATIENTS WITH CHRONIC HCV HAS CONSISTENTLY BEEN SHOWN TO BE ASSOCIATED WITH ANIMPAIRED RESPONSE TO ANTIVIRAL TREATMENT WITH INTERFERON THERAPY. IT IS NOT CLEARWHETHER THIS ASSOCIATION BETWEEN OVERWEIGHT OR OBESITY AND POOR RESPONSE TOANTIVIRAL THERAPY IS DUE TO STEATOSIS AND MIGHT NOT ONLY BE LINKED TO OBESITY.ACCUMULATING EVIDENCE SUGGESTS THAT STEATOSIS PLAYS A ROLE IN HCV-RELATEDFIBROSIS, AND SUPPORT FOR THIS ALSO COMES FROM STUDIES SHOWING THAT WEIGHTREDUCTION IN THESE PATIENTS LEADS NOT ONLY TO A DECREASE IN STEATOSIS BUT ALSOIMPROVEMENT IN FIBROSIS SCORE. |
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AN INCREASED PREVALENCE OF STEATOSIS IN PATIENTS WITH HCV IS WELL ESTABLISHED. MOSTSTUDIES HAVE REPORTED APPROXIMATELY 50% PREVALENCE OF STEATOSIS AMONG PATIENTSUNDERGOING A LIVER BIOPSY BECAUSE OF HCV. IN COMPARISON, AMONG PATIENTS WITH AUTOIMMUNE HEPATITIS AND HEPATITIS B (HBV), STEATOSIS IS NOT COMMONLY OBSERVED. HEPATITIS C VIRUS (HCV) INFECTION IS AN IMPORTANT RISK FACTOR FOR INSULIN RESISTANCE(IR). THE LATTER IS THE PATHOGENIC FOUNDATION UNDERLYING METABOLIC SYNDROME,STEATOSIS AND CIRRHOSIS, AND POSSIBLY HEPATOCELLULAR CARCINOMA (HCC). WHEREAS THEOVERALL PREVALENCE OF IR IS 10%-25% OF THE POPULATION, THE PREVALENCE IR IN HCVINFECTION REACHES FIGURES RANGING BETWEEN 30% TO 70%. OBESITY AND/OR STEATOSIS INPATIENTS WITH CHRONIC HCV HAS CONSISTENTLY BEEN SHOWN TO BE ASSOCIATED WITH ANIMPAIRED RESPONSE TO ANTIVIRAL TREATMENT WITH INTERFERON THERAPY. IT IS NOT CLEARWHETHER THIS ASSOCIATION BETWEEN OVERWEIGHT OR OBESITY AND POOR RESPONSE TOANTIVIRAL THERAPY IS DUE TO STEATOSIS AND MIGHT NOT ONLY BE LINKED TO OBESITY.ACCUMULATING EVIDENCE SUGGESTS THAT STEATOSIS PLAYS A ROLE IN HCV-RELATEDFIBROSIS, AND SUPPORT FOR THIS ALSO COMES FROM STUDIES SHOWING THAT WEIGHTREDUCTION IN THESE PATIENTS LEADS NOT ONLY TO A DECREASE IN STEATOSIS BUT ALSOIMPROVEMENT IN FIBROSIS SCORE.</description><identifier>ISSN: 2247-4455</identifier><identifier>EISSN: 2285-9632</identifier><language>eng</language><publisher>Targu-Jiu: Research and Science Today</publisher><subject>Alcohol ; Bacteriology ; Biopsy ; Diabetes ; General Reference Works ; Genotype & phenotype ; Health and medicine and law ; Hepatitis ; Hepatitis C ; Hepatology ; Infections ; Insulin resistance ; Interferon ; Liver cancer ; Liver diseases ; Medicine ; Metabolic syndrome ; Obesity ; Parasitology ; Patients ; Pharmacy ; Signal transduction ; Steatosis ; Therapy ; Weight control</subject><ispartof>Research and science today, 2016-03, Vol.11 (1), p.147-155</ispartof><rights>Copyright University Constantin Brancusi of Târgu-Jiu Mar 2016</rights><lds50>peer_reviewed</lds50><oa>free_for_read</oa><woscitedreferencessubscribed>false</woscitedreferencessubscribed></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Uhttps://www.ceeol.com//api/image/getissuecoverimage?id=picture_2016_30429.jpg</thumbnail><link.rule.ids>314,776,780</link.rule.ids></links><search><creatorcontrib>Ungureanu, Anca</creatorcontrib><creatorcontrib>Gaman, Alice Elena</creatorcontrib><creatorcontrib>Drocas, Andrei Ioan</creatorcontrib><creatorcontrib>Tieranu, Eugen</creatorcontrib><creatorcontrib>Dobritoiu, Maria</creatorcontrib><title>THE INFLUENCE OF STEATOSIS AND CONJUGATE FACTORS OF RESPONSE TO ANTIVIRAL TREATMENT IN CRONIC HEPATITIS C</title><title>Research and science today</title><addtitle>Research and Science Today</addtitle><description>HEPATITIS C VIRUS (HCV) IS A MAJOR CAUSE OF CHRONIC LIVER DISEASE WORLDWIDE. AN INCREASED PREVALENCE OF STEATOSIS IN PATIENTS WITH HCV IS WELL ESTABLISHED. MOSTSTUDIES HAVE REPORTED APPROXIMATELY 50% PREVALENCE OF STEATOSIS AMONG PATIENTSUNDERGOING A LIVER BIOPSY BECAUSE OF HCV. IN COMPARISON, AMONG PATIENTS WITH AUTOIMMUNE HEPATITIS AND HEPATITIS B (HBV), STEATOSIS IS NOT COMMONLY OBSERVED. HEPATITIS C VIRUS (HCV) INFECTION IS AN IMPORTANT RISK FACTOR FOR INSULIN RESISTANCE(IR). THE LATTER IS THE PATHOGENIC FOUNDATION UNDERLYING METABOLIC SYNDROME,STEATOSIS AND CIRRHOSIS, AND POSSIBLY HEPATOCELLULAR CARCINOMA (HCC). WHEREAS THEOVERALL PREVALENCE OF IR IS 10%-25% OF THE POPULATION, THE PREVALENCE IR IN HCVINFECTION REACHES FIGURES RANGING BETWEEN 30% TO 70%. OBESITY AND/OR STEATOSIS INPATIENTS WITH CHRONIC HCV HAS CONSISTENTLY BEEN SHOWN TO BE ASSOCIATED WITH ANIMPAIRED RESPONSE TO ANTIVIRAL TREATMENT WITH INTERFERON THERAPY. IT IS NOT CLEARWHETHER THIS ASSOCIATION BETWEEN OVERWEIGHT OR OBESITY AND POOR RESPONSE TOANTIVIRAL THERAPY IS DUE TO STEATOSIS AND MIGHT NOT ONLY BE LINKED TO OBESITY.ACCUMULATING EVIDENCE SUGGESTS THAT STEATOSIS PLAYS A ROLE IN HCV-RELATEDFIBROSIS, AND SUPPORT FOR THIS ALSO COMES FROM STUDIES SHOWING THAT WEIGHTREDUCTION IN THESE PATIENTS LEADS NOT ONLY TO A DECREASE IN STEATOSIS BUT ALSOIMPROVEMENT IN FIBROSIS SCORE.</description><subject>Alcohol</subject><subject>Bacteriology</subject><subject>Biopsy</subject><subject>Diabetes</subject><subject>General Reference Works</subject><subject>Genotype & phenotype</subject><subject>Health and medicine and law</subject><subject>Hepatitis</subject><subject>Hepatitis C</subject><subject>Hepatology</subject><subject>Infections</subject><subject>Insulin resistance</subject><subject>Interferon</subject><subject>Liver cancer</subject><subject>Liver diseases</subject><subject>Medicine</subject><subject>Metabolic syndrome</subject><subject>Obesity</subject><subject>Parasitology</subject><subject>Patients</subject><subject>Pharmacy</subject><subject>Signal transduction</subject><subject>Steatosis</subject><subject>Therapy</subject><subject>Weight control</subject><issn>2247-4455</issn><issn>2285-9632</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2016</creationdate><recordtype>article</recordtype><sourceid>REL</sourceid><sourceid>BENPR</sourceid><recordid>eNpFj81KAzEURgdRsGjfwEXA9UB-J8kyxEwbGZMyk7otnTQBS7Hase9vSgXv5ruLc8_HvalmGAtWy4bg28tOeU0pY_fVfJr2sIxoGsblrPoISwOsa7u1cdoA34IhGBX8YAeg3AvQ3r2uFyoY0CodfD9ckN4MK-8GA4IvULDvtlcdCH05fDMuFB_QvXdWg6VZqWBDkenH6i5vD1Oa_-VDFVoT9LLu_MJq1dWRSlZnTtiIMdzFtCUiQplJGkeR-C5SlLeMEEF2UPKcaTOSmCNGBGUpCONQoPLvQ_V81X6djt_nNP1s9sfz6bM0bhAXHFKBOSvU05WKKR0P_wiVDUKQ_AKbZFRw</recordid><startdate>20160301</startdate><enddate>20160301</enddate><creator>Ungureanu, Anca</creator><creator>Gaman, Alice Elena</creator><creator>Drocas, Andrei Ioan</creator><creator>Tieranu, Eugen</creator><creator>Dobritoiu, Maria</creator><general>Research and Science Today</general><general>University Constantin Brancusi of Târgu-Jiu</general><scope>AE2</scope><scope>BIXPP</scope><scope>REL</scope><scope>3V.</scope><scope>7X2</scope><scope>8FE</scope><scope>8FG</scope><scope>8FH</scope><scope>8FK</scope><scope>ABJCF</scope><scope>ABUWG</scope><scope>AEUYN</scope><scope>AFKRA</scope><scope>AIMQZ</scope><scope>ARAPS</scope><scope>ATCPS</scope><scope>AZQEC</scope><scope>BBNVY</scope><scope>BENPR</scope><scope>BGLVJ</scope><scope>BHPHI</scope><scope>BKSAR</scope><scope>BYOGL</scope><scope>CCPQU</scope><scope>D1I</scope><scope>DWQXO</scope><scope>GNUQQ</scope><scope>HCIFZ</scope><scope>JQ2</scope><scope>K7-</scope><scope>KB.</scope><scope>L6V</scope><scope>LIQON</scope><scope>LK8</scope><scope>M0K</scope><scope>M7P</scope><scope>M7S</scope><scope>P5Z</scope><scope>P62</scope><scope>PATMY</scope><scope>PCBAR</scope><scope>PDBOC</scope><scope>PIMPY</scope><scope>PQEST</scope><scope>PQQKQ</scope><scope>PQUKI</scope><scope>PRINS</scope><scope>PTHSS</scope><scope>PYCSY</scope></search><sort><creationdate>20160301</creationdate><title>THE INFLUENCE OF STEATOSIS AND CONJUGATE FACTORS OF RESPONSE TO ANTIVIRAL TREATMENT IN CRONIC HEPATITIS C</title><author>Ungureanu, Anca ; Gaman, Alice Elena ; Drocas, Andrei Ioan ; Tieranu, Eugen ; Dobritoiu, Maria</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c495-f735b220dcea38c09f3ebb8e7dc41fa53383d097ff46b3cfc2131f98357081963</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>2016</creationdate><topic>Alcohol</topic><topic>Bacteriology</topic><topic>Biopsy</topic><topic>Diabetes</topic><topic>General Reference Works</topic><topic>Genotype & phenotype</topic><topic>Health and medicine and law</topic><topic>Hepatitis</topic><topic>Hepatitis C</topic><topic>Hepatology</topic><topic>Infections</topic><topic>Insulin resistance</topic><topic>Interferon</topic><topic>Liver cancer</topic><topic>Liver diseases</topic><topic>Medicine</topic><topic>Metabolic syndrome</topic><topic>Obesity</topic><topic>Parasitology</topic><topic>Patients</topic><topic>Pharmacy</topic><topic>Signal transduction</topic><topic>Steatosis</topic><topic>Therapy</topic><topic>Weight control</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Ungureanu, Anca</creatorcontrib><creatorcontrib>Gaman, Alice Elena</creatorcontrib><creatorcontrib>Drocas, Andrei Ioan</creatorcontrib><creatorcontrib>Tieranu, Eugen</creatorcontrib><creatorcontrib>Dobritoiu, Maria</creatorcontrib><collection>Central and Eastern European Online Library (C.E.E.O.L.) 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AN INCREASED PREVALENCE OF STEATOSIS IN PATIENTS WITH HCV IS WELL ESTABLISHED. MOSTSTUDIES HAVE REPORTED APPROXIMATELY 50% PREVALENCE OF STEATOSIS AMONG PATIENTSUNDERGOING A LIVER BIOPSY BECAUSE OF HCV. IN COMPARISON, AMONG PATIENTS WITH AUTOIMMUNE HEPATITIS AND HEPATITIS B (HBV), STEATOSIS IS NOT COMMONLY OBSERVED. HEPATITIS C VIRUS (HCV) INFECTION IS AN IMPORTANT RISK FACTOR FOR INSULIN RESISTANCE(IR). THE LATTER IS THE PATHOGENIC FOUNDATION UNDERLYING METABOLIC SYNDROME,STEATOSIS AND CIRRHOSIS, AND POSSIBLY HEPATOCELLULAR CARCINOMA (HCC). WHEREAS THEOVERALL PREVALENCE OF IR IS 10%-25% OF THE POPULATION, THE PREVALENCE IR IN HCVINFECTION REACHES FIGURES RANGING BETWEEN 30% TO 70%. OBESITY AND/OR STEATOSIS INPATIENTS WITH CHRONIC HCV HAS CONSISTENTLY BEEN SHOWN TO BE ASSOCIATED WITH ANIMPAIRED RESPONSE TO ANTIVIRAL TREATMENT WITH INTERFERON THERAPY. IT IS NOT CLEARWHETHER THIS ASSOCIATION BETWEEN OVERWEIGHT OR OBESITY AND POOR RESPONSE TOANTIVIRAL THERAPY IS DUE TO STEATOSIS AND MIGHT NOT ONLY BE LINKED TO OBESITY.ACCUMULATING EVIDENCE SUGGESTS THAT STEATOSIS PLAYS A ROLE IN HCV-RELATEDFIBROSIS, AND SUPPORT FOR THIS ALSO COMES FROM STUDIES SHOWING THAT WEIGHTREDUCTION IN THESE PATIENTS LEADS NOT ONLY TO A DECREASE IN STEATOSIS BUT ALSOIMPROVEMENT IN FIBROSIS SCORE.</abstract><cop>Targu-Jiu</cop><pub>Research and Science Today</pub><tpages>9</tpages><oa>free_for_read</oa></addata></record> |
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| subjects | Alcohol Bacteriology Biopsy Diabetes General Reference Works Genotype & phenotype Health and medicine and law Hepatitis Hepatitis C Hepatology Infections Insulin resistance Interferon Liver cancer Liver diseases Medicine Metabolic syndrome Obesity Parasitology Patients Pharmacy Signal transduction Steatosis Therapy Weight control |
| title | THE INFLUENCE OF STEATOSIS AND CONJUGATE FACTORS OF RESPONSE TO ANTIVIRAL TREATMENT IN CRONIC HEPATITIS C |
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