Significance of Blood and Cerebrospinal Fluid Biomarkers for Alzheimer's Disease: Sensitivity, Specificity and Potential for Clinical Use von D'Abramo, Cristina, D'Adamio, Luciano, Giliberto, Luca

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Tau passive immunotherapy in mutant P301L mice: antibody affinity versus specificity von d'Abramo, Cristina, Acker, Christopher M, Jimenez, Heidy T, Davies, Peter

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Passive Immunization in JNPL3 Transgenic Mice Using an Array of Phospho-Tau Specific Antibodies von d'Abramo, Cristina, Acker, Christopher M, Jimenez, Heidy, Davies, Peter

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Multi-Drug Cocktail Therapy Improves Survival and Neurological Function after Asphyxial Cardiac Arrest in Rodents von Choudhary, Rishabh C, Shoaib, Muhammad, Hayashida, Kei, Yin, Tai, Miyara, Santiago J, d'Abramo, Cristina, Heuser, William G, Shinozaki, Koichiro, Kim, Nancy, Takegawa, Ryosuke, Nishikimi, Mitsuaki, Li, Timmy, Owens, Casey, Molmenti, Ernesto P, He, Mingzhu, Vanpatten, Sonya, Al-Abed, Yousef, Kim, Junhwan, Becker, Lance B

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Transgenic expression of the amyloid-beta precursor protein-intracellular domain does not induce Alzheimer's Disease-like traits in vivo von Giliberto, Luca, d'Abramo, Cristina, Acker, Christopher Michael, Davies, Peter, D'Adamio, Luciano

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Analysis of early effects of human APOE isoforms on Alzheimer's disease and type III hyperlipoproteinemia pathways using knock-in rat models with humanized APP and APOE von Yesiltepe, Metin, Yin, Tao, Tambini, Marc, Bao, Hanmei, Pan, Meixia, d'Abramo, Cristina, Giliberto, Luca, Han, Xianlin, Luciano, D'Adamio

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Anti-tau conformational scFv MC1 antibody efficiently reduces pathological tau species in adult JNPL3 mice von Vitale, Francesca, Giliberto, Luca, Ruiz, Santiago, Steslow, Kristen, Marambaud, Philippe, d'Abramo, Cristina

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Oxidative stress-induced MMP- and γ-secretase-dependent VE-cadherin processing is modulated by the proteasome and BMP9/10 von Ivaldo, Caterina, Passalacqua, Mario, Furfaro, Anna Lisa, d’Abramo, Cristina, Ruiz, Santiago, Chatterjee, Prodyot K., Metz, Christine N., Nitti, Mariapaola, Marambaud, Philippe

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Endogenous retroviruses are associated with hippocampus-based memory impairment von Sankowski, Roman, Strohl, Joshua J., Huerta, Tomás S., Nasiri, Elham, Mazzarello, Andrea N., D’Abramo, Cristina, Cheng, Kai Fan, Staszewski, Ori, Prinz, Marco, Huerta, Patricio T., Al-Abed, Yousef

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CB2 receptor deficiency increases amyloid pathology and alters tau processing in a transgenic mouse model of Alzheimer's disease von Koppel, Jeremy, Vingtdeux, Valerie, Marambaud, Philippe, d'Abramo, Cristina, Jimenez, Heidy, Stauber, Mark, Friedman, Rachel, Davies, Peter

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Novel synthetic small-molecule activators of AMPK as enhancers of autophagy and amyloid-β peptide degradation von Vingtdeux, Valérie, Chandakkar, Pallavi, Zhao, Haitian, d'Abramo, Cristina, Davies, Peter, Marambaud, Philippe

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Microarray Analysis in Alzheimer's Disease and Normal Aging von Ricciarelli, Roberta, d'Abramo, Cristina, Massone, Sara, Marinari, Umberto M., Pronzato, Maria A., Tabaton, Massimo

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Interferon regulatory factor 5 modulates microglia response to tau by interfering with phagocytosis von Giliberto, Luca, d'Abramo, Cristina, Cicalo, Julia, Hou, Jianwei, Matta, Bharati, Barnes, Betsy

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Intramuscular injection of vectorized-scFvMC1 reduces pathological tau in two different tau transgenic models von Vitale, Francesca, Ortolan, Jasmin, Volpe, Bruce T., Marambaud, Philippe, Giliberto, Luca, d'Abramo, Cristina

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Sensitive quantitative assays for tau and phospho-tau in transgenic mouse models von Acker, Christopher M, Forest, Stefanie K, Zinkowski, Ray, Davies, Peter, d'Abramo, Cristina

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Aβ43 levels determine the onset of pathological amyloid deposition von Tambini, Marc D., Yin, Tao, Yesiltepe, Metin, Breuillaud, Lionel, Zehntner, Simone P., d'Abramo, Cristina, Giliberto, Luca, D'Adamio, Luciano

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Detecting tau in serum of transgenic animal models after tau immunotherapy treatment von d'Abramo, Cristina, Acker, Christopher M, Schachter, Joel B, Terracina, Giuseppe, Wang, Xiaohai, Forest, Stefanie K, Davies, Peter

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CB₂ receptor deficiency increases amyloid pathology and alters tau processing in a transgenic mouse model of Alzheimer's disease von Koppel, Jeremy, Vingtdeux, Valerie, Marambaud, Philippe, d'Abramo, Cristina, Jimenez, Heidy, Stauber, Mark, Friedman, Rachel, Davies, Peter

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Memory deficits due to familial British dementia BRI2 mutation are caused by loss of BRI2 function rather than amyloidosis von Tamayev, Robert, Giliberto, Luca, Li, Wei, d'Abramo, Cristina, Arancio, Ottavio, Vidal, Ruben, D'Adamio, Luciano

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Erratum to: CB2 Receptor Deficiency Increases Amyloid Pathology and Alters Tau Processing in a Transgenic Mouse Model of Alzheimer’s Disease von Koppel, Jeremy, Vingtdeux, Valerie, Marambaud, Philippe, d’Abramo, Cristina, Jimenez, Heidy, Stauber, Mark, Friedman, Rachel, Davies, Peter

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