A review of non-probabilistic machine learning-based state of health estimation techniques for Lithium-ion battery von Sui, Xin, He, Shan, Vilsen, Søren B., Meng, Jinhao, Teodorescu, Remus, Stroe, Daniel-Ioan

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Battery state-of-health modelling by multiple linear regression von Vilsen, Søren B., Stroe, Daniel-Ioan

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Smart Battery Technology for Lifetime Improvement von Teodorescu, Remus, Sui, Xin, Vilsen, Søren B., Bharadwaj, Pallavi, Kulkarni, Abhijit, Stroe, Daniel-Ioan

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Transfer Learning for Adapting Battery State-of-Health Estimation From Laboratory to Field Operation von Vilsen, Soren B., Stroe, Daniel-Ioan

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On the Use of Randomly Selected Partial Charges to Predict Battery State-of-Health von Vilsen, Søren B., Stroe, Daniel-Ioan

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Dataset of lithium-ion battery degradation based on a forklift mission profile for state-of-health estimation and lifetime prediction von Vilsen, Søren B., Stroe, Daniel-Ioan

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Modelling allelic drop-outs in STR sequencing data generated by MPS von Vilsen, Søren B., Tvedebrink, Torben, Eriksen, Poul S., Hussing, Christian, Børsting, Claus, Morling, Niels

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Statistical modelling of Ion PGM HID STR 10-plex MPS Data von Vilsen, Søren B, Tvedebrink, Torben, Mogensen, Helle Smidt, Morling, Niels

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Stutter analysis of complex STR MPS data von Vilsen, Søren B., Tvedebrink, Torben, Eriksen, Poul Svante, Bøsting, Claus, Hussing, Christian, Mogensen, Helle Smidt, Morling, Niels

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Decreased neuronal Na+,K+‐ATPase activity in Atp1a3 heterozygous mice increases susceptibility to depression‐like endophenotypes by chronic variable stress von Kirshenbaum, G. S., Saltzman, K., Rose, B., Petersen, J., Vilsen, B., Roder, J. C.

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A C-terminal mutation of ATP1A3 underscores the crucial role of sodium affinity in the pathophysiology of rapid-onset dystonia-parkinsonism von Blanco-Arias, Patricia, Einholm, Anja P., Mamsa, Hafsa, Concheiro, Carla, Gutiérrez-de-Terán, Hugo, Romero, Jesús, Toustrup-Jensen, Mads S., Carracedo, Ángel, Jen, Joanna C., Vilsen, Bente, Sobrido, María-Jesús

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Mutant Glu781-->Ala of the rat kidney Na+,K(+)-ATPase displays low cation affinity and catalyzes ATP hydrolysis at a high rate in the absence of potassium ions von Vilsen, B

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Leucine 332 at the Boundary Between the Fourth Transmembrane Segment and the Cytoplasmic Domain of Na+,K+-ATPase Plays a Pivotal Role in the Ion Translocating Conformational Change... von Vilsen, Bente

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Structure-function relationships of cation translocation by Ca(2+)- and Na+, K(+)-ATPases studied by site-directed mutagenesis von Andersen, J P, Vilsen, B

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Genetic suppression of agrin reduces mania‐like behavior in Na+, K+‐ATPase α3 mutant mice von Kirshenbaum, G. S., Clapcote, S. J., Petersen, J., Vilsen, B., Ralph, M. R., Roder, J. C.

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Glutamate 329 located in the fourth transmembrane segment of the α-subunit of the rat kidney Na+,K+-ATPase is not an essential residue for active transport of sodium and potassium... von VILSEN, B

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Mutant Phe788 → Leu of the Na+,K+-ATPase Is Inhibited by Micromolar Concentrations of Potassium and Exhibits High Na+-ATPase Activity at Low Sodium Concentrations von Vilsen, Bente

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Functional consequences of alterations to Pro 328 and Leu 332 located in the 4th transmembrane segment of the α-subunit of the rat kidney Na +,K +-ATPase von Vilsen, Bente

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Transmembrane segment M3 is essential to thapsigargin sensitivity of the sarcoplasmic reticulum Ca(2+)-ATPase von Nørregaard, A, Vilsen, B, Andersen, J P

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