Morpholino-mediated SOD1 reduction ameliorates an amyotrophic lateral sclerosis disease phenotype von Nizzardo, M., Simone, C., Rizzo, F., Ulzi, G., Ramirez, A., Rizzuti, M., Bordoni, A., Bucchia, M., Gatti, S., Bresolin, N., Comi, G. P., Corti, S.

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The analysis of myotonia congenita mutations discloses functional clusters of amino acids within the CBS2 domain and the C‐terminal peptide of the ClC‐1 channel von Altamura, C, Lucchiari, S, Sahbani, D, Ulzi, G, Comi, GP, D'Ambrosio, P, Petillo, R, Politano, L, Vercelli, L, Mongini, T, Dotti, MT, Cardani, R, Meola, G, Lo Monaco, M, Matthews, E, Hanna, MG, Carratù, MR, Conte, D, Imbrici, P, Desaphy, J-F

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The analysis of myotonia congenita mutations discloses functional clusters of amino acids within the CBS2 domain and the C‐terminal peptide of the ClC‐1 channel von Altamura, Concetta, Lucchiari, Sabrina, Sahbani, Dalila, Ulzi, Gianna, Comi, Giacomo P., D'Ambrosio, Paola, Petillo, Roberta, Politano, Luisa, Vercelli, Liliana, Mongini, Tiziana, Dotti, Maria Teresa, Cardani, Rosanna, Meola, Giovanni, Lo Monaco, Mauro, Matthews, Emma, Hanna, Michael G., Carratù, Maria Rosaria, Conte, Diana, Imbrici, Paola, Desaphy, Jean‐François

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