PRDM15 is a key regulator of metabolism critical to sustain B-cell lymphomagenesis von Mzoughi, Slim, Fong, Jia Yi, Papadopoli, David, Koh, Cheryl M., Hulea, Laura, Pigini, Paolo, Di Tullio, Federico, Andreacchio, Giuseppe, Hoppe, Michal Marek, Wollmann, Heike, Low, Diana, Caldez, Matias J., Peng, Yanfen, Torre, Denis, Zhao, Julia N., Uchenunu, Oro, Varano, Gabriele, Motofeanu, Corina-Mihaela, Lakshmanan, Manikandan, Teo, Shun Xie, Wun, Cheng Mun, Perini, Giovanni, Tan, Soo Yong, Ong, Chee Bing, Al-Haddawi, Muthafar, Rajarethinam, Ravisankar, Hue, Susan Swee-Shan, Lim, Soon Thye, Ong, Choon Kiat, Huang, Dachuan, Ng, Siok-Bian, Bernstein, Emily, Hasson, Dan, Wee, Keng Boon, Kaldis, Philipp, Jeyasekharan, Anand, Dominguez-sola, David, Topisirovic, Ivan, Guccione, Ernesto

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Perturbations of cancer cell metabolism by the antidiabetic drug canagliflozin von Papadopoli, David, Uchenunu, Oro, Palia, Ranveer, Chekkal, Nabila, Hulea, Laura, Topisirovic, Ivan, Pollak, Michael, St-Pierre, Julie

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AMPK is required for recovery from metabolic stress induced by ultrasound microbubble treatment von Lo, Louis, Uchenunu, Oro, Botelho, Roberto J., Antonescu, Costin N., Karshafian, Raffi

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Mitochondrial complex IV defects induce metabolic and signaling perturbations that expose potential vulnerabilities in HCT116 cells von Uchenunu, Oro, Zhdanov, Alexander V., Hutton, Phillipe, Jovanovic, Predrag, Wang, Ye, Andreev, Dmitry E., Hulea, Laura, Papadopoli, David J., Avizonis, Daina, Baranov, Pavel V., Pollak, Michael N., Papkovsky, Dmitri B., Topisirovic, Ivan

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Oncogenic kinases and perturbations in protein synthesis machinery and energetics in neoplasia von Uchenunu, Oro, Pollak, Michael, Topisirovic, Ivan, Hulea, Laura

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Translational and HIF-1α-Dependent Metabolic Reprogramming Underpin Metabolic Plasticity and Responses to Kinase Inhibitors and Biguanides von Hulea, Laura, Gravel, Simon-Pierre, Morita, Masahiro, Cargnello, Marie, Uchenunu, Oro, Im, Young Kyuen, Lehuédé, Camille, Ma, Eric H., Leibovitch, Matthew, McLaughlan, Shannon, Blouin, Marie-José, Parisotto, Maxime, Papavasiliou, Vasilios, Lavoie, Cynthia, Larsson, Ola, Ohh, Michael, Ferreira, Tiago, Greenwood, Celia, Bridon, Gaëlle, Avizonis, Daina, Ferbeyre, Gerardo, Siegel, Peter, Jones, Russell G., Muller, William, Ursini-Siegel, Josie, St-Pierre, Julie, Pollak, Michael, Topisirovic, Ivan

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A hydride transfer complex reprograms NAD metabolism and bypasses senescence von Igelmann, Sebastian, Lessard, Frédéric, Uchenunu, Oro, Bouchard, Jacob, Fernandez-Ruiz, Ana, Rowell, Marie-Camille, Lopes-Paciencia, Stéphane, Papadopoli, David, Fouillen, Aurélien, Ponce, Katia Julissa, Huot, Geneviève, Mignacca, Lian, Benfdil, Mehdi, Kalegari, Paloma, Wahba, Haytham M., Pencik, Jan, Vuong, Nhung, Quenneville, Jordan, Guillon, Jordan, Bourdeau, Véronique, Hulea, Laura, Gagnon, Etienne, Kenner, Lukas, Moriggl, Richard, Nanci, Antonio, Pollak, Michael N., Omichinski, James G., Topisirovic, Ivan, Ferbeyre, Gerardo

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Abstract A20: Investigating the role of SCO2 in the metabolic adaptation of cancer cells von Zhdanov, Alexandre V., Uchenunu, Oro, Brazhe, Nadezda A., Nikelshparg, Evelina, Andreev, Dmitry E., Dmitriev, Ruslan I., Sosnovtseva, Olga V., Topisirovic, Ivan, Papkovsky, Dmitri B.

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Translational and HIF-1α-dependent metabolic reprogramming underpin metabolic plasticity and determine responses to kinase inhibitors and biguanides von Hulea, Laura, Gravel, Simon-Pierre, Morita, Masahiro, Cargnello, Marie, Uchenunu, Oro, Im, Young Kyuen, Lehuédé, Camille, Ma, Eric H., Leibovitch, Matthew, McLaughlan, Shannon, Blouin, Marie-José, Parisotto, Maxime, Papavasiliou, Vasilios, Lavoie, Cynthia, Larsson, Ola, Ohh, Michael, Ferreira, Tiago, Greenwood, Celia, Bridon, Gaëlle, Avizonis, Daina, Ferbeyre, Gerardo, Siegel, Peter, Jones, Russell G., Muller, William, Ursini-Siegel, Josie, St-Pierre, Julie, Pollak, Michael, Topisirovic, Ivan

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Mitochondrial complex IV defects induce metabolic and signaling perturbations that expose potential vulnerabilities in HCT116 cells von Oro Uchenunu

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Mitochondrial complex IV defects induce metabolic and signaling perturbations that expose potential vulnerabilities in HCT116 cells von Oro Uchenunu

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Mitochondrial complex IV defects induce metabolic and signaling perturbations that expose potential vulnerabilities in HCT116 cells von Oro Uchenunu

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A Hydride transfer complex reprograms NAD metabolism and bypasses senescence von Sebastian Igelmann

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A Hydride transfer complex reprograms NAD metabolism and bypasses senescence von Sebastian Igelmann

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