Neuronal deletion of Gtf2i, associated with Williams syndrome, causes behavioral and myelin alterations rescuable by a remyelinating drug von Barak, Boaz, Zhang, Zicong, Liu, Yuanyuan, Nir, Ariel, Trangle, Sari S., Ennis, Michaela, Levandowski, Kirsten M., Wang, Dongqing, Quast, Kathleen, Boulting, Gabriella L., Li, Yi, Bayarsaihan, Dashzeveg, He, Zhigang, Feng, Guoping

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Publisher Correction: Neuronal deletion of Gtf2i, associated with Williams syndrome, causes behavioral and myelin alterations rescuable by a remyelinating drug von Barak, Boaz, Zhang, Zicong, Liu, Yuanyuan, Nir, Ariel, Trangle, Sari S., Ennis, Michaela, Levandowski, Kirsten M., Wang, Dongqing, Quast, Kathleen, Boulting, Gabriella L., Li, Yi, Bayarsaihan, Dashzeveg, He, Zhigang, Feng, Guoping

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Hyperbaric Oxygen Therapy Alleviates Social Behavior Dysfunction and Neuroinflammation in a Mouse Model for Autism Spectrum Disorders von Fischer, Inbar, Shohat, Sophie, Levy, Gilad, Bar, Ela, Trangle, Sari Schokoroy, Efrati, Shai, Barak, Boaz

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Investigation of Different Molecular Weight Fucoidan Fractions Derived from New Zealand Undaria pinnatifida in Combination with GroA Therapy in Prostate Cancer Cell Lines von Yang, Xu, Wang, Sheng, Trangle, Sari Schokoroy, Li, Yan, White, William Lindsey, Li, Jinyao, Ying, Tianlei, Kong, Qingjun, Zhao, Yu, Lu, Jun

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In individuals with Williams syndrome, dysregulation of methylation in non-coding regions of neuronal and oligodendrocyte DNA is associated with pathology and cortical development von Trangle, Sari Schokoroy, Rosenberg, Tali, Parnas, Hadar, Levy, Gilad, Bar, Ela, Marco, Asaf, Barak, Boaz

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Neuronal Gtf2i deletion alters mitochondrial and autophagic properties von Nir Sade, Ariel, Levy, Gilad, Schokoroy Trangle, Sari, Elad Sfadia, Galit, Bar, Ela, Ophir, Omer, Fischer, Inbar, Rokach, May, Atzmon, Andrea, Parnas, Hadar, Rosenberg, Tali, Marco, Asaf, Elroy Stein, Orna, Barak, Boaz

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Nucleolin inhibitor GroA triggers reduction in epidermal growth factor receptor activation: Pharmacological implication for glial scarring after spinal cord injury von Goldshmit, Yona, Schokoroy Trangle, Sari, Afergan, Fabian, Iram, Tal, Pinkas‐Kramarski, Ronit

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Neuronal deletion of Gtf2i results in developmental microglial alterations in a mouse model related to Williams syndrome von Bar, Ela, Fischer, Inbar, Rokach, May, Elad‐Sfadia, Galit, Shirenova, Sophie, Ophir, Omer, Trangle, Sari Schokoroy, Okun, Eitan, Barak, Boaz

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Altered White Matter and microRNA Expression in a Murine Model Related to Williams Syndrome Suggests That miR-34b/c Affects Brain Development via Ptpru and Dcx Modulation von Grad, Meitar, Nir, Ariel, Levy, Gilad, Trangle, Sari Schokoroy, Shapira, Guy, Shomron, Noam, Assaf, Yaniv, Barak, Boaz

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Interfering with the interaction between ErbB1, nucleolin and Ras as a potential treatment for glioblastoma von Goldshmit, Yona, Trangle, Sari Schokoroy, Kloog, Yoel, Pinkas-Kramarski, Ronit

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Altered White Matter and microRNA Expression in a Murine Model Related to Williams Syndrome Suggests That miR-34b/c Affects Brain Development via P tpru and Dcx Modulation von Grad, Meitar, Nir, Ariel, Levy, Gilad, Trangle, Sari Schokoroy, Shapira, Guy, Shomron, Noam, Assaf, Yaniv, Barak, Boaz

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Enhancing FTS (Salirasib) efficiency via combinatorial treatment von Wolfson, Eya, Schmukler, Eran, Schokoroy, Sari Trangle, Kloog, Yoel, Pinkas-Kramarski, Ronit

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Shank3 mutation impairs glutamate signaling and myelination in ASD mouse model and human iPSC-derived OPCs von Fischer, Inbar, Shohat, Sophie, Leichtmann-Bardoogo, Yael, Nayak, Ritu, Wiener, Gal, Rosh, Idan, Shemen, Aviram, Tripathi, Utkarsh, Rokach, May, Bar, Ela, Hussein, Yara, Castro, Ana Carolina, Chen, Gal, Soffer, Adi, Schokoroy-Trangle, Sari, Elad-Sfadia, Galit, Assaf, Yaniv, Schroeder, Avi, Monteiro, Patricia, Stern, Shani, Maoz, Ben M, Barak, Boaz

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