Pervasive nuclear envelope ruptures precede ECM signaling and disease onset without activating cGAS-STING in Lamin-cardiomyopathy mice von En, Atsuki, Bogireddi, Hanumakumar, Thomas, Briana, Stutzman, Alexis V., Ikegami, Sachie, LaForest, Brigitte, Almakki, Omar, Pytel, Peter, Moskowitz, Ivan P., Ikegami, Kohta

Volltext

Advancements in mapping 3D genome architecture von McKay, Daniel J., Stutzman, Alexis V., Dowen, Jill M.

Volltext

Heterochromatic 3D genome organization is directed by HP1a- and H3K9-dependent and independent mechanisms von Stutzman, Alexis V., Hill, Christina A., Armstrong, Robin L., Gohil, Riya, Duronio, Robert J., Dowen, Jill M., McKay, Daniel J.

Volltext

Transcription-independent TFIIIC-bound sites cluster near heterochromatin boundaries within lamina-associated domains in C. elegans von Stutzman, Alexis V, Liang, April S, Beilinson, Vera, Ikegami, Kohta

Volltext

Abstract Tu086: Repairing nuclear envelope ruptures to ameliorate Lamin-related cardiomyopathy von En, Atsuki, Bogireddi, Hanumakumar, Thomas, Briana, Stutzman, Alexis, Ikegami, Sachie, Laforest, Brigitte, Almakki, Omar, Pytel, Peter, Moskowitz, Ivan, Ikegami, Kohta

Volltext

Abstract P3098: Immune Suppression Ameliorates A Mouse Model Of Lmna-related Cardiomyopathy With Nuclear Envelope Rupture von En, Atsuki, Bogireddi, Hanumakumar, Thomas, Briana, Stutzman, Alexis, Almakki, Omar, Ikegami, Sachie, Moskowitz, Ivan P, Ikegami, Kohta

Volltext

Abstract MP230: Nuclear Rupture In A Mouse Model Of Lmna-related Cardiomyopathy Causes Cytoplasmic Exposure Of The Proinflammatory Signaling Protein Hmgb1 von Ikegami, Kohta, Stutzman, Alexis, Ikegami, Sachie, Almakki, Omar, Liu, Sunny, Burnicka-turek, Ozanna, Moskowitz, Ivan

Volltext

Abstract 508: Nuclear Lamins At Enhancers: A New Hypothesis for Laminopathies von Ikegami, Kohta, Secchia, Stefano, Almakki, Omar, Stutzman, Alexis V, Ikegami, Sachie, Ayres, Natalia, Liu, Sunny Y, Lieb, Jason D, Moskowitz, Ivan P

Volltext

Heterochromatic 3D genome organization is directed by HP1a- and H3K9-dependent and independent mechanisms von Stutzman, Alexis

Volltext bestellen

Heterochromatic 3D genome organization is directed by HP1a- and H3K9-dependent and independent mechanisms von Stutzman, Alexis

Volltext bestellen

MOESM1 of Transcription-independent TFIIIC-bound sites cluster near heterochromatin boundaries within lamina-associated domains in C. elegans von Stutzman, Alexis, Liang, April, Beilinson, Vera, Kohta Ikegami

Volltext bestellen

Interrogating the role of the histone mark H3K9me3 in D. melanogaster Genome Organization and Gene Regulation von Stutzman, Alexis, Hill, Christina, Armstrong, Robin, Duronio, Robert, Dowen, Jill, McKay, Daniel J.

Volltext bestellen