Neuron loss in APP transgenic mice von Calhoun, Michael E, Wiederhold, Karl-Heinz, Abramowski, Dorothee, Phinney, Amie L, Probst, Alphonse, Sturchler-Pierrat, Christine, Staufenbiel, Matthias, Sommer, Bernd, Jucker, Mathias

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Spatial learning, exploration, anxiety, and motor coordination in female APP23 transgenic mice with the Swedish mutation von Lalonde, R, Dumont, M, Staufenbiel, M, Sturchler-Pierrat, C, Strazielle, C

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Progressive age-related impairment of cognitive behavior in APP23 transgenic mice von Kelly, P.H, Bondolfi, L, Hunziker, D, Schlecht, H.-P, Carver, K, Maguire, E, Abramowski, D, Wiederhold, K.-H, Sturchler-Pierrat, C, Jucker, M, Bergmann, R, Staufenbiel, M, Sommer, B

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The evolution of A beta peptide burden in the APP23 transgenic mice: implications for A beta deposition in Alzheimer disease von Kuo, Y M, Beach, T G, Sue, L I, Scott, S, Layne, K J, Kokjohn, T A, Kalback, W M, Luehrs, D C, Vishnivetskaya, T A, Abramowski, D, Sturchler-Pierrat, C, Staufenbiel, M, Weller, R O, Roher, A E

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Dietary Cu Stabilizes Brain Superoxide Dismutase 1 Activity and Reduces Amyloid Aβ Production in APP23 Transgenic Mice von Bayer, Thomas A., Schäfer, Stephanie, Simons, Andreas, Kemmling, André, Kamer, Thomas, Tepest, Ralf, Eckert, Anne, Schüssel, Katrin, Eikenberg, Oliver, Sturchler-Pierrat, Christine, Abramowski, Dorothee, Staufenbiel, Matthias, Multhaup, Gerd

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Regional brain cytochrome oxidase activity in β-amyloid precursor protein transgenic mice with the swedish mutation von STRAZIELLE, C, STURCHLER-PIERRAT, C, STAUFENBIEL, M, LALONDE, R

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Pathogenic Mechanisms of Alzheimer's Disease Analyzed in the APP23 Transgenic Mouse Model von STURCHLER-PIERRAT, CHRISTINE, STAUFENBIEL, MATTHIAS

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Aβ is targeted to the vasculature in a mouse model of hereditary cerebral hemorrhage with amyloidosis von Jucker, Mathias, Herzig, Martin C, Winkler, David T, Burgermeister, Patrick, Pfeifer, Michelle, Kohler, Esther, Schmidt, Stephen D, Danner, Simone, Abramowski, Dorothee, Stürchler-Pierrat, Christine, Bürki, Kurt, van Duinen, Sjoerd G, Maat-Schieman, Marion L C, Staufenbiel, Matthias, Mathews, Paul M

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Two Amyloid Precursor Protein Transgenic Mouse Models with Alzheimer Disease-Like Pathology von Sturchler-Pierrat, Christine, Abramowski, Dorothee, Duke, Mairead, Wiederhold, Karl-Heinz, Mistl, Claudia, Rothacher, Sabin, Ledermann, Brigit, Bürki, Kurt, Frey, Peter, Paganetti, Paolo A., Waridel, Caroline, Calhoun, Michael E., Jucker, Mathias, Probst, Alphonse, Staufenbiel, Matthias, Sommer, Bernd

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Neuronal Overexpression of Mutant Amyloid Precursor Protein Results in Prominent Deposition of Cerebrovascular Amyloid von Calhoun, Michael E., Burgermeister, Patrick, Phinney, Amie L., Stalder, Martina, Tolnay, Markus, Wiederhold, Karl-Heinz, Abramowski, Dorothee, Sturchler-Pierrat, Christine, Sommer, Bernd, Staufenbiel, Matthias, Jucker, Mathias

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RNAs mediating cotranslational insertion of selenocysteine in eukaryotic selenoproteins von Hubert, N, Walczak, R, Sturchler, C, Myslinski, E, Schuster, C, Westhof, E, Carbon, P, Krol, A

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Compromised Hemodynamic Response in Amyloid Precursor Protein Transgenic Mice von Mueggler, Thomas, Sturchler-Pierrat, Christine, Baumann, Diana, Rausch, Martin, Staufenbiel, Matthias, Rudin, Markus

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Comparative analysis of amyloid-beta chemical structure and amyloid plaque morphology of transgenic mouse and Alzheimer's disease brains von Kuo, Y M, Kokjohn, T A, Beach, T G, Sue, L I, Brune, D, Lopez, J C, Kalback, W M, Abramowski, D, Sturchler-Pierrat, C, Staufenbiel, M, Roher, A E

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Impaired hypoxic tolerance and altered protein binding of NADH in presymptomatic APP23 transgenic mice von Büchner, M, Huber, R, Sturchler-Pierrat, C, Staufenbiel, M, Riepe, M.W

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Destabilization of β-catenin by mutations in presenilin-1 potentiates neuronal apoptosis von Yankner, Bruce A, Zhang, Zhuohua, Hartmann, Henrike, Minh Do, Viet, Abramowski, Dorothee, Sturchler-Pierrat, Christine, Staufenbiel, Matthias, Sommer, Bernd, van de Wetering, Marc, Clevers, Hans, Saftig, Paul, De Strooper, Bart, He, Xi

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Expression of human β‐secretase in the mouse brain increases the steady‐state level of β‐amyloid von Bodendorf, Ursula, Danner, Simone, Fischer, Frauke, Stefani, Muriel, Sturchler‐Pierrat, Christine, Wiederhold, Karl‐Heinz, Staufenbiel, Matthias, Paganetti, Paolo

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Amyloid β interacts with the amyloid precursor protein: a potential toxic mechanism in Alzheimer's disease von Yankner, Bruce A, Lorenzo, Alfredo, Yuan, Menglan, Zhang, Zhuohua, Paganetti, Paolo A, Sturchler-Pierrat, Christine, Staufenbiel, Matthias, Mautino, Jorge, Vigo, Francisco Sol, Sommer, Bernd

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Cholinergic Changes in the APP23 Transgenic Mouse Model of Cerebral Amyloidosis von Boncristiano, Sonia, Calhoun, Michael E, Kelly, Peter H, Pfeifer, Michelle, Bondolfi, Luca, Stalder, Martina, Phinney, Amie L, Abramowski, Dorothee, Sturchler-Pierrat, Christine, Enz, Albert, Sommer, Bernd, Staufenbiel, Matthias, Jucker, Mathias

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Selenocysteylation in eukaryotes necessitates the uniquely long aminoacyl acceptor stem of selenocysteine tRNA(Sec) von Sturchler-Pierrat, C. (Institut de Biologie Moleculaire et Cellulaire, Strasbourg, France.), Hubert, N, Totsuka, T, Mizutani, T, Carbon, P, Krol, A

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The 9/4 secondary structure of eukaryotic selenocysteine tRNA: More pieces of evidence von HUBERT, NADIA, STURCHLER, CHRISTINE, WESTHOF, ERIC, CARBON, PHILIPPE, KROL, ALAIN

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