IL-10 Deficiency Reveals a Role for TLR2-Dependent Bystander Activation of T Cells in Lyme Arthritis von Whiteside, Sarah K, Snook, Jeremy P, Ma, Ying, Sonderegger, F Lynn, Fisher, Colleen, Petersen, Charisse, Zachary, James F, Round, June L, Williams, Matthew A, Weis, Janis J

Volltext

Autologous cancer cell vaccination, adoptive T‐cell transfer, and interleukin‐2 administration results in long‐term survival for companion dogs with osteosarcoma von Flesner, Brian K., Wood, Gary W., Gayheart‐Walsten, Pamela, Sonderegger, F. Lynn, Henry, Carolyn J., Tate, Deborah J., Bechtel, Sandra M., Donnelly, Lindsay L., Johnson, Gayle C., Kim, Dae Young, Wahaus, Tammie A., Bryan, Jeffrey N., Reyes, Noe

Volltext

The immune response to picornavirus infection and the effect of immune manipulation on acute seizures von DePaula-Silva, Ana Beatriz, Sonderegger, F. Lynn, Libbey, Jane E., Doty, Daniel J., Fujinami, Robert S.

Volltext

Localized production of IL-10 suppresses early inflammatory cell infiltration and subsequent development of IFN-γ-mediated Lyme arthritis von Sonderegger, F Lynn, Ma, Ying, Maylor-Hagan, Heather, Brewster, James, Huang, Xiaosong, Spangrude, Gerald J, Zachary, James F, Weis, John H, Weis, Janis J

Volltext

Endothelial cells and fibroblasts amplify the arthritogenic type I IFN response in murine Lyme disease and are major sources of chemokines in Borrelia burgdorferi-infected joint ti... von Lochhead, Robert B, Sonderegger, F Lynn, Ma, Ying, Brewster, James E, Cornwall, Doug, Maylor-Hagen, Heather, Miller, Jennifer C, Zachary, James F, Weis, John H, Weis, Janis J

Volltext

TLR2-dependent bystander activation of T cells results in exacerbation of Lyme arthritis von Whiteside, Sarah, Snook, Jeremy P., Ma, Ying, Sonderegger, F. Lynn, Fisher, Colleen, Petersen, Charisse, Zachary, James F., Round, June L., Williams, Matthew A., Weis, Janis J.

Volltext

The IL-10-/- mouse as a model of chronic Lyme arthritis. (169.16) von Sonderegger, F. Lynn, Ma, Ying, Weis, John, Weis, Janis

Volltext

Endothelial cells and fibroblasts amplify the arthritogenic Type I IFN response in murine Lyme Disease and are major sources of chemokines in B. burgdorferi-infected joint tissue1 von Lochhead, Robert B., Sonderegger, F. Lynn, Ma, Ying, Brewster, James E., Cornwall, Doug, Maylor-Hagen, Heather, Miller, Jennifer C., Zachary, James F., Weis, John H., Weis, Janis J.

Volltext

Localized Production of IL-10 Suppresses Early Inflammatory Cell Infiltration and Subsequent Development of IFN-γ-Mediated Lyme Arthritis1 von Sonderegger, F. Lynn, Ma, Ying, Maylor-Hagan, Heather, Brewster, James, Huang, Xiaosong, Spangrude, Gerald J., Zachary, James F., Weis, John H., Weis, Janis J.

Volltext

Needleless luer access port von LYNN LAWRENCE A, HARDING WESTON F, SONDEREGGER RALPH L

Volltext bestellen

Needleless luer access port von Harding, Weston F, Lynn, Lawrence A, Sonderegger, Ralph L

Volltext bestellen

Needleless luer access port von Harding, Weston F, Lynn, Lawrence A, Sonderegger, Ralph L

Volltext bestellen

Needleless luer access port von LYNN LAWRENCE A, HARDING WESTON F, SONDEREGGER RALPH L

Volltext bestellen