Dominantly Inherited Alzheimer Network Trials Unit (DIAN-TU): Trial Satisfaction and Attitudes towards Future Clinical Trials von Liu, Haiyan, Li, J., Ziegemeier, E., Adams, S., McDade, E., Clifford, D. B., Cao, Y., Wang, G., Li, Y., Mills, S. L., Santacruz, A. M., Belyew, S., Grill, J. D., Snider, B. J., Mummery, C. J., Surti, G., Hannequin, D., Wallon, D., Berman, S. B., Jimenez-Velazquez, I. Z., Roberson, E. D., van Dyck, C. H., Honig, L. S., Sanchez-Valle, R., Brooks, W. S., Gauthier, S., Galasko, D., Masters, C. L., Brosch, J., Hsiung, G.-Y. R., Jayadev, S., Formaglio, M., Masellis, M., Clarnette, R., Pariente, J., Dubois, B., Pasquier, F., Bateman, R. J., Llibre-Guerra, Jorge J.

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Neurons derived from embryonic stem (ES) cells resemble normal neurons in their vulnerability to excitotoxic death von Qu, Y, Vadivelu, S, Choi, L, Liu, S, Lu, A, Lewis, B, Girgis, R, Lee, C.S, Snider, B.J, Gottlieb, D.I, McDonald, J.W

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Neocortical neurons cultured from mice with expanded cag repeats in the huntingtin gene: unaltered vulnerability to excitotoxins and other insults von Snider, B.J, Moss, J.L, Revilla, F.J, Lee, C.-S, Wheeler, V.C, Macdonald, M.E, Choi, D.W

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Nitric oxide reduces Ca2+ and Zn2+ influx through voltage-gated Ca2+ channels and reduces Zn2+ neurotoxicity von SNIDER, B. J, CHOI, J, TURETSKY, D. M, CANZONIERO, L. M. T, SENSI, S. L, SHELINE, C. T, WANG, X, YU, S. P, CHOI, D. W

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P4-063 Very early onset familial Alzheimer's disease with lewy bodies without known causative mutations von Snider, B.J., Hou, Craig E., Jervis, Ramiro, Lendon, Corinne L., Norton, JoAnne, Goate, Alison M., McKeel, Dan W., Morris, John C.

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Nitric oxide reduces Ca 2+ and Zn 2+ influx through voltage-gated Ca 2+ channels and reduces Zn 2+ neurotoxicity von Snider, B.J, Choi, J, Turetsky, D.M, Canzoniero, L.M.T, Sensi, S.L, Sheline, C.T, Wang, X, Yu, S.P, Choi, D.W

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Possible new class II alleles in African-Americans revealed by sequence specific oligonucleotide probes von Schmeckpeper, B.J., Robbins, F., Margolet, L., Snider, T., White, B., Bonadonna, M., Bias, W.B.

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