Apoptosis is regulated by the VDAC1 N-terminal region and by VDAC oligomerization: release of cytochrome c, AIF and Smac/Diablo von Shoshan-Barmatz, Varda, Keinan, Nurit, Abu-Hamad, Salah, Tyomkin, Dalia, Aram, Lior

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The Expression Level of the Voltage-Dependent Anion Channel Controls Life and Death of the Cell von Abu-Hamad, Salah, Sivan, Sara, Shoshan-Barmatz, Varda

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Key regions of VDAC1 functioning in apoptosis induction and regulation by hexokinase von Shoshan-Barmatz, Varda, Zakar, Miri, Rosenthal, Keshet, Abu-Hamad, Salah

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Endogenous macrophage migration inhibitory factor reduces the accumulation and toxicity of misfolded SOD1 in a mouse model of ALS von Leyton-Jaimes, Marcel F., Benaim, Clara, Abu-Hamad, Salah, Kahn, Joy, Guetta, Amos, Bucala, Richard, Israelson, Adrian

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Targeting the Mitochondrial Protein VDAC1 as a Potential Therapeutic Strategy in ALS von Shteinfer-Kuzmine, Anna, Argueti-Ostrovsky, Shirel, Leyton-Jaimes, Marcel F., Anand, Uttpal, Abu-Hamad, Salah, Zalk, Ran, Shoshan-Barmatz, Varda, Israelson, Adrian

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Macrophage Migration Inhibitory Factor as a Chaperone Inhibiting Accumulation of Misfolded SOD1 von Israelson, Adrian, Ditsworth, Dara, Sun, Shuying, Song, SungWon, Liang, Jason, Hruska-Plochan, Marian, McAlonis-Downes, Melissa, Abu-Hamad, Salah, Zoltsman, Guy, Shani, Tom, Maldonado, Marcus, Bui, Anh, Navarro, Michael, Zhou, Huilin, Marsala, Martin, Kaspar, Brian K., Da Cruz, Sandrine, Cleveland, Don W.

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VDAC1 N-terminus is essential both for apoptosis and the protective effect of anti-apoptotic proteins von Abu-Hamad, Salah, Arbel, Nir, Calo, Doron, Arzoine, Laetitia, Israelson, Adrian, Keinan, Nurit, Ben-Romano, Ronit, Friedman, Orr, Shoshan-Barmatz, Varda

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MIF inhibits the formation and toxicity of misfolded SOD1 amyloid aggregates: implications for familial ALS von Shvil, Neta, Banerjee, Victor, Zoltsman, Guy, Shani, Tom, Kahn, Joy, Abu-Hamad, Salah, Papo, Niv, Engel, Stanislav, Bernhagen, Jurgen, Israelson, Adrian

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A VDAC1-Derived N-Terminal Peptide Inhibits Mutant SOD1-VDAC1 Interactions and Toxicity in the SOD1 Model of ALS von Shteinfer-Kuzmine, Anna, Argueti, Shirel, Gupta, Rajeev, Shvil, Neta, Abu-Hamad, Salah, Gropper, Yael, Hoeber, Jan, Magrì, Andrea, Messina, Angela, Kozlova, Elena N, Shoshan-Barmatz, Varda, Israelson, Adrian

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Localization of the voltage-dependent anion channel-1 Ca2+ -binding sites von Israelson, Adrian, Abu-Hamad, Salah, Zaid, Hilal, Nahon, Edna, Shoshan-Barmatz, Varda

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An N-terminal nucleotide-binding site in VDAC1: Involvement in regulating mitochondrial function von Yehezkel, Galit, Abu-Hamad, Salah, Shoshan-Barmatz, Varda

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Mapping the ruthenium red-binding site of the voltage-dependent anion channel-1 von Israelson, Adrian, Zaid, Hilal, Abu-Hamad, Salah, Nahon, Edna, Shoshan-Barmatz, Varda

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Hexokinase-I Protection against Apoptotic Cell Death Is Mediated via Interaction with the Voltage-dependent Anion Channel-1 von Abu-Hamad, Salah, Zaid, Hilal, Israelson, Adrian, Nahon, Edna, Shoshan-Barmatz, Varda

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In self-defence: hexokinase promotes voltage-dependent anion channel closure and prevents mitochondria-mediated apoptotic cell death von Azoulay-Zohar, Heftsi, Israelson, Adrian, Abu-Hamad, Salah, Shoshan-Barmatz, Varda

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Hexokinase-I Protection against Apoptotic Cell Death Is Mediated via Interaction with the Voltage-dependent Anion Channel-1: MAPPING THE SITE OF BINDING von Abu-Hamad, Salah, Zaid, Hilal, Israelson, Adrian, Nahon, Edna, Shoshan-Barmatz, Varda

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Fluoxetine (Prozac) interaction with the mitochondrial voltage-dependent anion channel and protection against apoptotic cell death von Nahon, Edna, Israelson, Adrian, Abu-Hamad, Salah, Shoshan-Barmatz, Varda

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Misfolded SOD1 Accumulation and Mitochondrial Association Contribute to the Selective Vulnerability of Motor Neurons in Familial ALS: Correlation to Human Disease von Abu-Hamad, Salah, Kahn, Joy, Leyton-Jaimes, Marcel F, Rosenblatt, Jonathan, Israelson, Adrian

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Macrophage migration inhibitory factor as a component of selective vulnerability of motor neurons in ALS von Abu-Hamad, Salah, Israelson, Adrian

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Dominant-negative VDAC1 mutants reveal oligomeric VDAC1 to be the active unit in mitochondria-mediated apoptosis von Mader, Asaf, Abu-Hamad, Salah, Arbel, Nir, Gutiérrez-Aguilar, Manuel, Gutierr-Asuilar, Manuel, Shoshan-Barmatz, Varda

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Localization of the voltage-dependent anion channel-1 Ca super(2+)-binding sites von Israelson, Adrian, Abu-Hamad, Salah, Zaid, Hilal, Nahon, Edna, Shoshan-Barmatz, Varda

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