FLT3 mutations in acute myeloid leukemia cell lines von Quentmeier, H, Reinhardt, J, Zaborski, M, Drexler, H G

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The NPM1 wild-type OCI-AML2 and the NPM1-mutated OCI-AML3 cell lines carry DNMT3A mutations von Tiacci, E, Spanhol-Rosseto, A, Martelli, M P, Pasqualucci, L, Quentmeier, H, Grossmann, V, Drexler, H G, Falini, B

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EZH2 Y641 mutations in follicular lymphoma von Bödör, C, O'Riain, C, Wrench, D, Matthews, J, Iyengar, S, Tayyib, H, Calaminici, M, Clear, A, Iqbal, S, Quentmeier, H, Drexler, H G, Montoto, S, Lister, A T, Gribben, J G, Matolcsy, A, Fitzgibbon, J

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MARIMO cells harbor a CALR mutation but are not dependent on JAK2/STAT5 signaling von Kollmann, K, Nangalia, J, Warsch, W, Quentmeier, H, Bench, A, Boyd, E, Scott, M, Drexler, H G, Green, A R

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Fusion of NUP214 to ABL1 on amplified episomes in T-cell acute lymphoblastic leukemia von Graux, C., Cools, J., Melotte, C., Quentmeier, H., Ferrando, A., Levine, R., Vermeesch, J.R., Stul, M., Dutta, B., Boeckx, N., Bosly, A., Heimann, P., Uyttebroeck, A., Mentens, N., Somers, R., MacLeod, R.A.F., Drexler, H.G., Look, A.T., Gilliland, D.G., Michaux, L., Vandenberghe, P., Wlodarska, I., Marynen, Peter, Hagemeijer, Anne

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JAK2 V617F tyrosine kinase mutation in cell lines derived from myeloproliferative disorders von QUENTMEIER, H, MACLEOD, R. A. F, ZABORSKI, M, DREXLER, H. G

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Cloning of human thymic stromal lymphopoietin (TSLP) and signaling mechanisms leading to proliferation von QUENTMEIER, H, DREXLER, H. G, FLECKENSTEIN, D, ZABORSKI, M, ARMSTRONG, A, SIMS, J. E, LYMAN, S. D

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U-2932: two clones in one cell line, a tool for the study of clonal evolution von Quentmeier, H, Amini, R M, Berglund, M, Dirks, W G, Ehrentraut, S, Geffers, R, MacLeod, R A F, Nagel, S, Romani, J, Scherr, M, Zaborski, M, Drexler, H G

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SOCS2: inhibitor of JAK2V617F-mediated signal transduction von Quentmeier, H, Geffers, R, Jost, E, MacLeod, R A F, Nagel, S, Röhrs, S, Romani, J, Scherr, M, Zaborski, M, Drexler, H G

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Amplification at 7q22 targets cyclin-dependent kinase 6 in T-cell lymphoma von Nagel, S, Leich, E, Quentmeier, H, Meyer, C, Kaufmann, M, Drexler, H G, Zettl, A, Rosenwald, A, MacLeod, R A F

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MEF2C is activated by multiple mechanisms in a subset of T-acute lymphoblastic leukemia cell lines von Nagel, S, Meyer, C, Quentmeier, H, Kaufmann, M, Drexler, H G, MacLeod, R A F

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Chromosome 11q23 aberrations activating FOXR1 in B-cell lymphoma von Pommerenke, C, Hauer, V, Zaborski, M, MacLeod, R A F, Nagel, S, Amini, R M, Berglund, M, Geffers, R, Drexler, H G, Quentmeier, H

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A novel ABL1 fusion to the SH2 containing inositol phosphatase-1 (SHIP1) in acute lymphoblastic leukemia (ALL) von Kakadia, P M, Tizazu, B, Mellert, G, Harbott, J, Röttgers, S, Quentmeier, H, Spiekermann, K, Bohlander, S K

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Malignant hematopoietic cell lines: in vitro models for the study of MLL gene alterations von Drexler, H G, Quentmeier, H, MacLeod, R A F

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Epigenetic regulation of brain expressed X-linked-2, a marker for acute myeloid leukemia with mixed lineage leukemia rearrangements von Fischer, C, Drexler, H G, Reinhardt, J, Zaborski, M, Quentmeier, H

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Cell line OCI/AML3 bears exon-12 NPM gene mutation-A and cytoplasmic expression of nucleophosmin von QUENTMEIER, H, MARTELLI, M. P, MARTELLI, M. F, MECUCCI, C, DREXLER, H. G, FALINI, B, DIRKS, W. G, BOLLI, N, LISO, A, MACLEOD, R. A. F, NICOLETTI, I, MANNUCCI, R, PUCCIARINI, A, BIGERNA, B

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Hypermethylation of Death-Associated Protein Kinase 1 differentiates natural killer cell lines from cell lines derived from T-acute lymphoblastic leukemia von Röhrs, S, Romani, J, Zaborski, M, Drexler, H G, Quentmeier, H

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HLXB9 activates IL6 in Hodgkin lymphoma cell lines and is regulated by PI3K signalling involving E2F3 von Nagel, S, Scherr, M, Quentmeier, H, Kaufmann, M, Zaborski, M, Drexler, H G, MacLeod, R A F

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Expression of BEX1 in acute myeloid leukemia with MLL rearrangements von Quentmeier, H, Tonelli, R, Geffers, R, Pession, A, Uphoff, C C, Drexler, H G

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e6-a2 BCR-ABL1 fusion in T-cell acute lymphoblastic leukemia von Quentmeier, H, Cools, J, MacLeod, R A F, Marynen, P, Uphoff, C C, Drexler, H G

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