Cardiac Pacemaker Activity and Aging von Peters, Colin H, Sharpe, Emily J, Proenza, Catherine

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Depolarization of the conductance-voltage relationship in the NaV1.5 mutant, E1784K, is due to altered fast inactivation von Peters, Colin H, Yu, Alec, Zhu, Wandi, Silva, Jonathan R, Ruben, Peter C

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Regulation of HCN Channels by Protein Interactions von Peters, Colin H, Singh, Rohit K, Bankston, John R, Proenza, Catherine

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Isoform-specific regulation of HCN4 channels by a family of endoplasmic reticulum proteins von Peters, Colin H., Myers, Mallory E., Juchno, Julie, Haimbaugh, Charlie, Bichraoui, Hicham, Du, Yanmei, Bankston, John R., Walker, Lori A., Proenza, Catherine

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Defining the interactions between HCN4 and the regulatory proteins IRAG and LRMP von Singh, Rohit K., Langley, Avery A., Peters, Colin H., Bankston, John, Proenza, Cathy

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Functional characterization of a metal ion coordination site in HCN4 von Blecker, Lucas M., Peters, Colin H., Peraza, Diego A., Bankston, John, Proenza, Cathy

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Calcium inhibits the funny current (If)in sinoatrial myocytes von Peters, Colin H., Peraza, Diego A., Blecker, Lucas M., Bankston, John, Proenza, Cathy

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Differential thermosensitivity in mixed syndrome cardiac sodium channel mutants von Abdelsayed, Mena, Peters, Colin H., Ruben, Peter C.

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Bidirectional flow of the funny current (If) during the pacemaking cycle in murine sinoatrial node myocytes von Peters, Colin H., Liu, Pin W., Morotti, Stefano, Gantz, Stephanie C., Grandi, Eleonora, Bean, Bruce P., Proenza, Catherine

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The funny current If is essential for the fight-or-flight response in cardiac pacemaker cells von Peters, Colin H, Rickert, Christian, Morotti, Stefano, Grandi, Eleonora, Aronow, Kurt A, Beam, Kurt G, Proenza, Catherine

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Intracellular Na + Modulates Pacemaking Activity in Murine Sinoatrial Node Myocytes: An In Silico Analysis von Morotti, Stefano, Ni, Haibo, Peters, Colin H, Rickert, Christian, Asgari-Targhi, Ameneh, Sato, Daisuke, Glukhov, Alexey V, Proenza, Catherine, Grandi, Eleonora

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LRMP inhibits cAMP potentiation of HCN4 channels by disrupting intramolecular signal transduction von Peters, Colin H, Singh, Rohit K, Langley, Avery A, Nichols, William G, Ferris, Hannah R, Jeffrey, Danielle A, Proenza, Catherine, Bankston, John R

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E1784K, the most common Brugada syndrome and long-QT syndrome type 3 mutant, disrupts sodium channel inactivation through two separate mechanisms von Peters, Colin H, Watkins, Abeline R, Poirier, Olivia L, Ruben, Peter C

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Triggers for arrhythmogenesis in the Brugada and long QT 3 syndromes von Peters, Colin H., Abdelsayed, Mena, Ruben, Peter C.

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Functional Genomics of Epilepsy and Associated Neurodevelopmental Disorders Using Simple Animal Models: From Genes, Molecules to Brain Networks von Rosch, Richard, Burrows, Dominic R W, Jones, Laura B, Peters, Colin H, Ruben, Peter, Samarut, Éric

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Case studies in neuroscience: a novel amino acid duplication in the NH2-terminus of the brain sodium channel NaV1.1 underlying Dravet syndrome von Angus, Madeline, Peters, Colin H, Poburko, Damon, Brimble, Elise, Spelbrink, Emily M, Ruben, Peter C

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Case studies in neuroscience: a novel amino acid duplication in the NH 2 -terminus of the brain sodium channel Na V 1.1 underlying Dravet syndrome von Angus, Madeline, Peters, Colin H, Poburko, Damon, Brimble, Elise, Spelbrink, Emily M, Ruben, Peter C

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The L1624Q Variant in SCN1A Causes Familial Epilepsy Through a Mixed Gain and Loss of Channel Function von Jones, Laura B, Peters, Colin H, Rosch, Richard E, Owers, Maxine, Hughes, Elaine, Pal, Deb K, Ruben, Peter C

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A Mixed Periodic Paralysis & Myotonia Mutant, P1158S, Imparts pH-Sensitivity in Skeletal Muscle Voltage-gated Sodium Channels von Ghovanloo, Mohammad-Reza, Abdelsayed, Mena, Peters, Colin H., Ruben, Peter C.

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Effects of acidosis on neuronal voltage-gated sodium channels: Nav1.1 and Nav1.3 von Ghovanloo, Mohammad-Reza, Peters, Colin H., Ruben, Peter C.

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