Ha-ras activates the Na+/H+ antiporter by a protein kinase C-independent mechanism von Maly, K, Überall, F, Loferer, H, Doppler, W, Oberhuber, H, Groner, B, Grunicke, H H

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Mechanism of desensitization of the Ca2(+)-mobilizing system to bombesin by Ha-ras. Independence from down-modulation of agonist-stimulated inositol phosphate production von OBERHUBER, H, MALY, K, UÊBERALL, F, HOFLACHER, J, KIANI, A, GRUNICKE, H. H

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Hexadecylphosphocholine inhibits inositol phosphate formation and protein kinase C activity von Uberall, F, Oberhuber, H, Maly, K, Zaknun, J, Demuth, L, Grunicke, H H

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Topical Fk506 in inflammatory corneal and conjunctival diseases. A pilot study von Reinhard, Thomas, Reis, Alexander, Mayweg, Susanne, Oberhuber, Hermann, Mathis, Georg, Sundmacher, Rainer

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Desensitization of the Ca2+-mobilizing system to serum growth factors by Ha-ras and v-mos von MALY, K, DOPPLER, W, OBERHUBER, H, MEUSBURGER, H, HOFMANN, J, JAGGI, R, GRUNICKE, H. H

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Partial restoration of pre-transformation levels of lysyl oxidase and transin mRNAs in phenotypic ras revertants von Oberhuber, Hermann, Seliger, Barbara, Schäfer, Reinhold

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Functional interaction of wild-type and mutant p53 transfected into human tumor cell lines carrying activated RAS genes von SANJAI SHARMA, SCHWARTE-WALDHOFF, I, OBERHUBER, H, SCHÄFER, R

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The phospholipid- and calcium-dependent protein kinase as a target in tumor chemotherapy von Grunicke, H., Hofmann, J., Maly, K., Überall, F., Posch, L., Oberhuber, H., Fiebig, H.

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Interference of Ha-ras with inositol trisphosphate-mediated Ca(2+)-release von Maly, K, Kiani, A, Oberhuber, H, Grunicke, H

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Interference of Ha‐ras with inositol trisphosphate‐mediated Ca2+‐release von Maly, Karl, Kiani, Alexander, Oberhuber, Hermann, Grunicke, Hans

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Mechanism and biological significance of the Ha- ras-induced activation of the Na +/H +-antiporter von Maly, K., Hochleitner, B., Überall, F., Loferer, H., Oberhuber, H., Doppler, W., Grunicke, H.

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Effect of Ha-ras on phosphatidylinositol metabolism, Na+/H+-antiporter and mobilization of intracellular calcium von Maly, K, Oberhuber, H, Doppler, W, Hoflacher, J, Jaggi, R, Groner, B, Grunicke, H

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Effect of Ha-ras on mitogen-induced Ca(2+)- and K(+)-fluxes von Grunicke, H, Maly, K, Meyer, M, Oberhuber, H, Bichler, I, Waldegger, L S, Woell, E, Lang, F

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Interference of Ha-ras with inositol trisphosphate-mediated Ca super(2+)-release von Maly, K, Kiani, A, Oberhuber, H, Grunicke, H

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Plasma membrane as target of alkylating agents von Grunicke, H., Doppler, W., Hofmann, J., Lindner, H., Maly, K., Oberhuber, H., Ringsdorf, H., Roberts, J.J.

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Mechanism of desensitization of the Ca super(2+)-mobilizing system to bombesin by Ha-ras: Independence from down-modulation of agonist-stimulated inositol phosphate production von Oberhuber, H, Maly, K, Ueberall, F, Hoflacher, J, Kiani, A, Grunicke, H H

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Hexadecylphosphocholine inhibits inositols phosphate formation and protein kinase activity von UÊBERALL, F, OBERHUBER, H, MALY, K, ZAKNUM, J, DEMUTH, L, GRUNICKE, H. H

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Ha-ras activates the Na super(+)/H super(+) antiporter by a protein kinase C-independent mechanism von Maly, K, Ueberall, F, Loferer, H, Doppler, W, Oberhuber, H, Groner, B, Grunicke, H H

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Desensitization of the Ca super(2+)-mobilizing system to serum growth factors by Ha-ras and v-mos von Maly, K, Doppler, W, Oberhuber, H, Meusburger, H, Hofmann, J, Jaggi, R, Grunicke, H H

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Nitrogen mustard interference with potassium transport systems in Ehrlich ascites tumor cells von Doppler, W, Hofmann, J, Oberhuber, H, Maly, K, Grunicke, H

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