Beyond the Channel: Metabotropic Signaling by Nicotinic Receptors von Kabbani, Nadine, Nichols, Robert A.

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Selective coactivation of α7- and α4β2-nicotinic acetylcholine receptors reverses beta-amyloid–induced synaptic dysfunction von Roberts, Jessica P., Stokoe, Sarah A., Sathler, Matheus F., Nichols, Robert A., Kim, Seonil

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The neuroprotective N-terminal amyloid-β core hexapeptide reverses reactive gliosis and gliotoxicity in Alzheimer's disease pathology models von Lantz, Megan J, Roberts, Alyssa M, Delgado, Donovan D, Nichols, Robert A

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Editorial: Beta Amyloid: From Physiology to Pathogenesis von Nichols, Robert A, Gulisano, Walter, Puzzo, Daniela

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The Neuroprotective Beta Amyloid Hexapeptide Core Reverses Deficits in Synaptic Plasticity in the 5xFAD APP/PS1 Mouse Model von Forest, Kelly H., Taketa, Ruth, Arora, Komal, Todorovic, Cedomir, Nichols, Robert A.

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Assessing Neuroprotective Agents for Aβ-Induced Neurotoxicity von Forest, Kelly H., Nichols, Robert A.

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Neuroprotective amyloid β N‐terminal peptides differentially alter human α7‐ and α7β2‐nicotinic acetylcholine (nACh) receptor single‐channel properties von Roberts, Catherine F., Cao, Yiwei, Im, Wonpil, Nichols, Robert A., Lukas, Ronald J., George, Andrew A.

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Nicotinic Acetylcholine Receptors Sensitize a MAPK-linked Toxicity Pathway on Prolonged Exposure to β-Amyloid von Arora, Komal, Cheng, Justin, Nichols, Robert A.

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Isoorientin, a GSK-3β inhibitor, rescues synaptic dysfunction, spatial memory deficits and attenuates pathological progression in APP/PS1 model mice von Tan, Xiaoqin, Liang, Zhibin, Li, Yingui, Zhi, Yingkun, Yi, Lang, Bai, Shasha, Forest, Kelly H., Nichols, Robert A., Dong, Yan, Li, Qing X

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Theft Is Property: Dispossession and Critical Theory von Nichols, Robert

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Anti-Neuroinflammatory Effects of a Semi-Synthetic Isoorientin-Based Glycogen Synthase Kinase-3β Inhibitor in Lipopolysaccharide-Activated Microglial Cells von Xu, Meng, Lantz, Megan J, Nichols, Robert A, Li, Qing X

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Defense Communications: APL’s Contributions Through the Decades von Nichols, Robert A

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Transcriptome Profile of Nicotinic Receptor-Linked Sensitization of Beta Amyloid Neurotoxicity von Arora, Komal, Belcaid, Mahdi, Lantz, Megan J., Taketa, Ruth, Nichols, Robert A.

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Regulation of presynaptic Ca2+, synaptic plasticity and contextual fear conditioning by a N-terminal β-amyloid fragment von Lawrence, James L M, Tong, Mei, Alfulaij, Naghum, Sherrin, Tessi, Contarino, Mark, White, Michael M, Bellinger, Frederick P, Todorovic, Cedomir, Nichols, Robert A

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JIP1-Mediated JNK Activation Negatively Regulates Synaptic Plasticity and Spatial Memory von Morel, Caroline, Sherrin, Tessi, Kennedy, Norman J, Forest, Kelly H, Avcioglu Barutcu, Seda, Robles, Michael, Carpenter-Hyland, Ezekiel, Alfulaij, Naghum, Standen, Claire L, Nichols, Robert A, Benveniste, Morris, Davis, Roger J, Todorovic, Cedomir

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Serotonin, presynaptic 5‐HT3 receptors and synaptic plasticity in the developing cerebellum von Nichols, Robert A.

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Protection against β‐amyloid neurotoxicity by a non‐toxic endogenous N‐terminal β‐amyloid fragment and its active hexapeptide core sequence von Forest, Kelly H., Alfulaij, Naghum, Arora, Komal, Taketa, Ruth, Sherrin, Tessi, Todorovic, Cedomir, Lawrence, James L. M., Yoshikawa, Gene T., Ng, Ho‐Leung, Hruby, Victor J., Nichols, Robert A.

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beta -Amyloid activates presynaptic alpha 7 nicotinic acetylcholine receptors reconstituted into a model nerve cell system: involvement of lipid rafts von Khan, Ghous M, Tong, Mei, Jhun, Michelle, Arora, Komal, Nichols, Robert A

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Impact of Sustained Exposure to β-Amyloid on Calcium Homeostasis and Neuronal Integrity in Model Nerve Cell System Expressing α4β2 Nicotinic Acetylcholine Receptors von Arora, Komal, Alfulaij, Naghum, Higa, Jason K., Panee, Jun, Nichols, Robert A.

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A Constitutive, Transient Receptor Potential-like Ca2+ Influx Pathway in Presynaptic Nerve Endings Independent of Voltage-gated Ca2+ Channels and Na+/Ca2+ Exchange von Nichols, Robert A., Dengler, Andrew F., Nakagawa, Emily M., Bashkin, Marisa, Paul, Brian T., Wu, Jianlin, Khan, Ghous M.

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