Sialylation of IgG Fc domain impairs complement-dependent cytotoxicity von Quast, Isaak, Keller, Christian W, Maurer, Michael A, Giddens, John P, Tackenberg, Björn, Wang, Lai-Xi, Münz, Christian, Nimmerjahn, Falk, Dalakas, Marinos C, Lünemann, Jan D

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Elevated Epstein-Barr virus-encoded nuclear antigen-1 immune responses predict conversion to multiple sclerosis von Lünemann, Jan D., Tintoré, Mar, Messmer, Brady, Strowig, Till, Rovira, Álex, Perkal, Héctor, Caballero, Estrella, Münz, Christian, Montalban, Xavier, Comabella, Manuel

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Regulation of antibody effector functions through IgG Fc N-glycosylation von Quast, Isaak, Peschke, Benjamin, Lünemann, Jan D.

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Fc-Galactosylation of Human Immunoglobulin Gamma Isotypes Improves C1q Binding and Enhances Complement-Dependent Cytotoxicity von Peschke, Benjamin, Keller, Christian W, Weber, Patrick, Quast, Isaak, Lünemann, Jan D

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Targeting sialylation to treat central nervous system diseases von Lünemann, Jan D., von Gunten, Stephan, Neumann, Harald

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TNF-related apoptosis inducing ligand (TRAIL) as a potential response marker for interferon-beta treatment in multiple sclerosis von Wandinger, Klaus-Peter, Lünemann, Jan D, Wengert, Oliver, Bellmann-Strobl, Judith, Aktas, Orhan, Weber, Alexandra, Grundström, Eva, Ehrlich, Stefan, Wernecke, Klaus-D, Volk, Hans-Dieter, Zipp, Frauke

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Attenuated immune control of Epstein–Barr virus in humanized mice is associated with the multiple sclerosis risk factor HLA‐DR15 von Zdimerova, Hana, Murer, Anita, Engelmann, Christine, Raykova, Ana, Deng, Yun, Gujer, Cornelia, Rühl, Julia, McHugh, Donal, Caduff, Nicole, Naghavian, Reza, Pezzino, Gaetana, Capaul, Riccarda, Zbinden, Andrea, Ferlazzo, Guido, Lünemann, Jan D., Martin, Roland, Chatterjee, Bithi, Münz, Christian

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Neonatal Fc Receptor–Targeted Therapies in Neurology von Nelke, Christopher, Spatola, Marianna, Schroeter, Christina B., Wiendl, Heinz, Lünemann, Jan D.

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Antiviral immune responses: triggers of or triggered by autoimmunity? von Münz, Christian, Lünemann, Jan D, Getts, Meghann Teague, Miller, Stephen D

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ATG-dependent phagocytosis in dendritic cells drives myelin-specific CD4⁺ T cell pathogenicity during CNS inflammation von Keller, Christian W., Sina, Christina, Kotur, Monika B., Ramelli, Giulia, Mundt, Sarah, Quast, Isaak, Ligeon, Laure-Anne, Weber, Patrick, Becher, Burkhard, Münz, Christian, Lünemann, Jan D.

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Multiple Sclerosis Therapy Consensus Group (MSTCG): position statement on disease-modifying therapies for multiple sclerosis (white paper) von Wiendl, Heinz, Gold, Ralf, Berger, Thomas, Derfuss, Tobias, Linker, Ralf, Mäurer, Mathias, Aktas, Orhan, Baum, Karl, Berghoff, Martin, Bittner, Stefan, Chan, Andrew, Czaplinski, Adam, Deisenhammer, Florian, Di Pauli, Franziska, Du Pasquier, Renaud, Enzinger, Christian, Fertl, Elisabeth, Gass, Achim, Gehring, Klaus, Gobbi, Claudio, Goebels, Norbert, Guger, Michael, Haghikia, Aiden, Hartung, Hans-Peter, Heidenreich, Fedor, Hoffmann, Olaf, Kallmann, Boris, Kleinschnitz, Christoph, Klotz, Luisa, Leussink, Verena I., Leutmezer, Fritz, Limmroth, Volker, Lünemann, Jan D., Lutterotti, Andreas, Meuth, Sven G., Meyding-Lamadé, Uta, Platten, Michael, Rieckmann, Peter, Schmidt, Stephan, Tumani, Hayrettin, Weber, Frank, Weber, Martin S., Zettl, Uwe K., Ziemssen, Tjalf, Zipp, Frauke

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Skeletal muscle provides the immunological micro-milieu for specific plasma cells in anti-synthetase syndrome-associated myositis von Preuße, Corinna, Paesler, Barbara, Nelke, Christopher, Cengiz, Derya, Müntefering, Thomas, Roos, Andreas, Amelin, Damien, Allenbach, Yves, Uruha, Akinori, Dittmayer, Carsten, Hentschel, Andreas, Pawlitzki, Marc, Hoffmann, Sarah, Timm, Sara, Louis, Sarah Leonard, Dengler, Nora F., Wiendl, Heinz, Lünemann, Jan D., Sickmann, Albert, Hervier, Baptiste, Meuth, Sven G., Schneider, Udo, Schänzer, Anne, Krause, Sabine, Tomaras, Stylianos, Feist, Eugen, Hasseli, Rebecca, Goebel, Hans-Hilmar, Gallay, Laure, Streichenberger, Nathalie, Benveniste, Olivier, Stenzel, Werner, Ruck, Tobias

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Endocytosis regulation by autophagy proteins in MHC restricted antigen presentation von .Keller, Christian W, Loi, Monica, Ligeon, Laure-Anne, Gannagé, Monique, Lünemann, Jan D, Münz, Christian

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Fc Glycan-Modulated Immunoglobulin G Effector Functions von Quast, Isaak, Lünemann, Jan D.

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Complement activation contributes to GAD antibody-associated encephalitis von Chuquisana, Omar, Strippel, Christine, Tröscher, Anna M., Baumgartner, Tobias, Rácz, Attila, Keller, Christian W., Elger, Christian E., Melzer, Nico, Kovac, Stjepana, Wiendl, Heinz, Bauer, Jan, Lünemann, Jan D.

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Complement Activation Is a Prominent Feature of MOGAD von Keller, Christian W., Lopez, Joseph A., Wendel, Eva‐Maria, Ramanathan, Sudarshini, Gross, Catharina C., Klotz, Luisa, Reindl, Markus, Dale, Russell C., Wiendl, Heinz, Rostásy, Kevin, Brilot, Fabienne, Lünemann, Jan D.

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β-Amyloid is a substrate of autophagy in sporadic inclusion body myositis von Lünemann, Jan D., Schmidt, Jens, Schmid, Dorothee, Barthel, Konstanze, Wrede, Arne, Dalakas, Marinos C., Münz, Christian

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Antibody-mediated cell depletion therapies in multiple sclerosis von Mariottini, Alice, Muraro, Paolo A., Lünemann, Jan D.

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NEUROMUSCULAR DISEASE Getting specific: targeting Fc receptors in myasthenia gravis von Lunemann, Jan D.

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EBNA1-specific T cells from patients with multiple sclerosis cross react with myelin antigens and co-produce IFN-gamma and IL-2 von Lünemann, Jan D, Jelcić, Ilijas, Roberts, Susanne, Lutterotti, Andreas, Tackenberg, Björn, Martin, Roland, Münz, Christian

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