Multimodular biosensors reveal a novel platform for activation of G proteins by growth factor receptors von Midde, Krishna K., Aznar, Nicolas, Laederich, Melanie B., Ma, Gary S., Kunkel, Maya T., Newton, Alexandra C., Ghosh, Pradipta

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The Leucine-rich Repeat Protein LRIG1 Is a Negative Regulator of ErbB Family Receptor Tyrosine Kinases von Laederich, Melanie B, Funes-Duran, Melanie, Yen, Lily, Ingalla, Ellen, Wu, Xiuli, Carraway, 3rd, Kermit L, Sweeney, Colleen

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Achondroplasia: pathogenesis and implications for future treatment von Laederich, Melanie B, Horton, William A

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FGFR3 targeting strategies for achondroplasia von Laederich, Melanie B., Horton, William A.

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Sprouty 2 disturbs FGFR3 degradation in thanatophoric dysplasia type II: A severe form of human achondroplasia von Guo, Changsheng, Degnin, Catherine R., Laederich, Melanie B., Lunstrum, Gregory P., Holden, Paul, Bihlmaier, Jeanie, Krakow, Deborah, Cho, Yoon-Jae, Horton, William A.

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Ligand activation leads to regulated intramembrane proteolysis of fibroblast growth factor receptor 3 von Degnin, Catherine R, Laederich, Melanie B, Horton, William A

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Fibroblast growth factor receptor 3 (FGFR3) is a strong heat shock protein 90 (Hsp90) client: implications for therapeutic manipulation von Laederich, Melanie B, Degnin, Catherine R, Lunstrum, Gregory P, Holden, Paul, Horton, William A

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FGFs in endochondral skeletal development von Degnin, Catherine R., Laederich, Melanie B., Horton, William A.

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Fibroblast Growth Factor Receptor 3 (FGFR3) Is a Strong Heat Shock Protein 90 (Hsp90) Client von Laederich, Melanie B., Degnin, Catherine R., Lunstrum, Gregory P., Holden, Paul, Horton, William A.

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The leucine-rich repeat protein LRIG1 is a negative regulator of ErbB family receptor tyrosine kinases. Vol. 279 (2004) 47050–47056 von Laederich, Melanie B., Funes-Duran, Melanie, Yen, Lily, Ingalla, Ellen, Wu, Xiuli, Carraway, Kermit L., Sweeney, Colleen

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Treatment for achondroplasia von HORTON WILLIAM A, LAEDERICH MELANIE B

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TREATMENT FOR ACHONDROPLASIA von HORTON WILLIAM A, LAEDERICH MELANIE B

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