Hypertonic Stress Increases T Cell Interleukin-2 Expression through a Mechanism That Involves ATP Release, P2 Receptor, and p38 MAPK Activation von Loomis, William H, Namiki, Sachiko, Ostrom, Rennolds S, Insel, Paul A, Junger, Wolfgang G

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Hypertonicity rescues T cells from suppression by trauma-induced anti-inflammatory mediators von Loomis, William H, Namiki, Sachiko, Hoyt, David B, Junger, Wolfgang G

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Hypertonic saline infusion : Can it regulate human neutrophil function? von ANGLE, Niren, CABELLO-PASSINI, Ruy, HOYT, David B, LOOMIS, William H, SHREVE, Amy, NAMIKI, Sachiko, JUNGER, Wolfgang G

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Hypertonic saline resuscitation restores hemorrhage-induced immunosuppression by decreasing prostaglandin E2 and interleukin-4 production von COIMBRA, R, JUNGER, W. G, HOYT, D. B, LIU, F. C, LOOMIS, W. H, EVERS, M. F

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Acute lung injury in endotoxemic rats is associated with sustained circulating IL-6 levels and intrapulmonary CINC activity and neutrophil recruitment--role of circulating TNF-alph... von Simons, R K, Junger, W G, Loomis, W H, Hoyt, D B

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Effects of trauma on immune cell function: impairment of intracellular calcium signaling von Hoyt, D B, Junger, W G, Loomis, W H, Liu, F C

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Tumor necrosis factor antibody treatment of septic baboons reduces the production of sustained T-cell suppressive factors von Junger, W G, Hoyt, D B, Redl, H, Liu, F C, Loomis, W H, Davies, J, Schlag, G

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Granulocytes and Hyperoxia Act Synergistically in Causing Acute Lung Injury von Krieger, Bruce P., Loomis, William H., Spragg, Roger G.

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Hypertonicity increases cAMP in PMN and blocks oxidative burst by PKA-dependent and -independent mechanisms von Orlic, Tatjana, Loomis, William H, Shreve, Amy, Namiki, Sachiko, Junger, Wolfgang G

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EFFECT OF HYPERTONIC SALINE ON T-LYMPHOCYTE FUNCTION: 469 von Junger, W. G., Chen, Y., Shukla, A., Hashiguchi, N., Loomis, W. H., Hoyt, D. B.

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Moonshine strategy and other stories von Hawks, Wells

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Hypertonic/hyperoncotic fluids reverse prostaglandin E2 (PGE2)-induced T-cell suppression von Coimbra, R, Junger, W G, Liu, F C, Loomis, W H, Hoyt, D B

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Lactated Ringer's Is Superior to Normal Saline in a Model of Massive Hemorrhage and Resuscitation von Healey, Mark A., Davis, Richard E., Liu, Forrest C., Loomis, William H., Hoyt, David B.

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Alteration in Ca2+ homeostasis by a trauma peptide von Hoyt, D B, Ozkan, A N, Frevert, J, Junger, W G, Loomis, W H

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Irreversible Shock Is Not Irreversible: A New Model of Massive Hemorrhage and Resuscitation von Healey, Mark A., Samphire, John, Hoyt, David B., Liu, Forrest, Davis, Richard, Loomis, William H.

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225 TNF ANTIBODY TREATMENT REDUCES THE PRODUCTION OF ACTIVE TGF-B1, BUT NOT OF IL-10 IN A BABOON SEPSIS MODEL von Junger, W. G., Hoyt, D. B., Redl, H., Liu, F. C., Loomis, W. H., Davies, J., Schlag, G.

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Immunosuppression after Endotoxin Shock: The Result of Multiple Anti-inflammatory Factors von Junger, Wolfgang G., Hoyt, David B., Liu, Forrest C., Loomis, William H., Coimbra, Raul

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Alteration in Ca1+ homeostasis by a trauma peptide von HOYT, D. B, NURI OZKAN, A, FREVERT, J, JUNGER, W. G, LOOMIS, W. H

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Inhibition of Enteral Enzymes by Enteroclysis with Nafamostat Mesilate Reduces Neutrophil Activation and Transfusion Requirements after Hemorrhagic Shock von Doucet, Jay J., Hoyt, David B., Coimbra, Raul, Schmid-Schönbein, Geert W., Junger, Wolfgang G., Wolf, Paul L., Loomis, William H., Hugli, Tony E.

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Immunosuppression after endotoxin shock : The result of multiple anti-inflammatory factors. Discussion von JUNGER, W. G, HOYT, D. B, LIU, F. C, LOOMIS, W. H, COIMBRA, R, CHEADLE, W. G

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