Gene therapy with a promoter targeting both rods and cones rescues retinal degeneration caused by AIPL1 mutations von SUN, X, PAWLYK, B, ALI, R. R, LI, T, XU, X, LIU, X, BULGAKOV, O. V, ADAMIAN, M, SANDBERG, M. A, KHANI, S. C, TAN, M.-H, SMITH, A. J

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Retraction Note: Gene therapy with a promoter targeting both rods and cones rescues retinal degeneration caused by AIPL1 mutations von Sun, X, Pawlyk, B, Xu, X, Liu, X, Bulgakov, O V, Adamian, M, Sandberg, M A, Khani, S C, Tan, M -H, Smith, A J, Ali, R R, Li, T

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Induction and tissue-specific expression of rabbit cytochrome P450IIE1 and IIE2 genes von PORTER, T. D, KHANI, S. C, COON, M. J

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Null Mutation in the Rhodopsin Kinase Gene Slows Recovery Kinetics of Rod and Cone Phototransduction in Man von Cideciyan, Artur V., Zhao, Xinyu, Nielsen, Lori, Khani, Shahrokh C., Jacobson, Samuel G., Palczewski, Krzysztof

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cDNA and Derived Amino Acid Sequence of Ethanol-Inducible Rabbit Liver Cytochrome P-450 Isozyme 3a (P-450ALC) von Khani, Shahrokh C., Zaphiropoulos, Peter G., Fujita, Valerie S., Porter, Todd D., Koop, Dennis R., Coon, Minor J.

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Molecular Forms of Human Rhodopsin Kinase (GRK1) von Zhao, X, Huang, J, Khani, S C, Palczewski, K

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Characterization and Chromosomal Localization of the Gene for Human Rhodopsin Kinase von Khani, Shahrokh C., Abitbol, Marc, Yamamoto, Shuji, Maravic-Magovcevic, Ivana, Dryja, Thaddeus P.

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Organization and differential expression of two highly similar genes in the rabbit alcohol-inducible cytochrome P-450 subfamily von Khani, S C, Porter, T D, Fujita, V S, Coon, M J

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Evaluation of the Rhodopsin Kinase Gene in Patients with Retinitis Pigmentosa von YAMAMOTO, SHUJI, KHANI, SHAHROKH C, BERSON, ELIOT L, DRYJA, THADDEUS P

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Replacement Gene Therapy with a Human RPGRIP1 Sequence Slows Photoreceptor Degeneration in a Murine Model of Leber Congenital Amaurosis von PAWLYK, Basil S, BULGAKOV, Oleg V, XIAOQING LIU, XIAOYUN XU, ADAMIAN, Michael, XUN SUN, KHANI, Shahrokh C, BERSON, Eliot L, SANDBERG, Michael A, TIANSEN LI

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Posterior segment intraocular foreign bodies von Khani, S C, Mukai, S

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Biochemical Evidence for Pathogenicity of Rhodopsin Kinase Mutations Correlated with the Oguchi Form of Congenital Stationary Night Blindness von Khani, Shahrokh C., Nielsen, Lori, Vogt, Todd M.

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Isolation and partial characterization of the gene for cytochrome P-450 3a (P-450 ALC) and a second closely related gene von Khani, Shahrokh C., Porter, Todd D., Coon, Minor J.

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Conserved interactions of a compact highly active enhancer/promoter upstream of the rhodopsin kinase (GRK1) gene von Young, Joyce E., Kasperek, Eileen M., Vogt, Todd M., Lis, Agnieszka, Khani, Shahrokh C.

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RETRACTED ARTICLE: Gene therapy with a promoter targeting both rods and cones rescues retinal degeneration caused by AIPL1 mutations von Sun, X, Pawlyk, B, Xu, X, Liu, X, Bulgakov, O V, Adamian, M, Sandberg, M A, Khani, S C, Tan, M-H, Smith, A J, Ali, R R, Li, T

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Expression characteristics of dual-promoter lentiviral vectors targeting retinal photoreceptors and Mueller cells von Semple-Rowland, S L, Coggin, W E, Geesey, M, Eccles, K S, Abraham, L, Pachigar, K, Ludlow, R, Khani, S C, Smith, W C

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11-cis retinol dehydrogenase mutations as a major cause of the congenital night-blindness disorder known as fundus albipunctatus von Gonzalez-Fernandez, F, Kurz, D, Bao, Y, Newman, S, Conway, B P, Young, J E, Han, D P, Khani, S C

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A Short, Highly Active Photoreceptor-Specific Enhancer/Promoter Region Upstream of the Human Rhodopsin Kinase Gene von Young, Joyce E, Vogt, Todd, Gross, Kenneth W, Khani, Shahrokh C

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Late-Onset Autosomal Dominant Macular Dystrophy with Choroidal Neovascularization and Nonexudative Maculopathy Associated with Mutation in the RDS Gene von Khani, Shahrokh C, Karoukis, Athanasios J, Young, Joyce E, Ambasudhan, Rajesh, Burch, Tracy, Stockton, Richard, Lewis, Richard Alan, Sullivan, Lori S, Daiger, Stephen P, Reichel, Elias, Ayyagari, Radha

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Conserved structure and spatiotemporal function of the compact rhodopsin kinase (GRK1) enhancer/promoter von Young, Joyce E, Gross, Kenneth W, Khani, Shahrokh C

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